...
机译:日本血吸虫可溶性蛋抗原诱导凋亡并抑制肝星状细胞的激活:一种可能的分子机制
Department of Pathogen Biology School of Medicine Nantong University 19 Qixiu Road Nantong;
Department of Pathogen Biology School of Medicine Nantong University 19 Qixiu Road Nantong;
Department of Pathogen Biology School of Medicine Nantong University 19 Qixiu Road Nantong;
Department of Pathogen Biology School of Medicine Nantong University 19 Qixiu Road Nantong;
Department of Pathogen Biology School of Medicine Nantong University 19 Qixiu Road Nantong;
Department of Pathogen Biology School of Medicine Nantong University 19 Qixiu Road Nantong;
Department of Pathogen Biology School of Medicine Nantong University 19 Qixiu Road Nantong;
Department of Pathogen Biology School of Medicine Nantong University 19 Qixiu Road Nantong;
Department of Pathogen Biology School of Medicine Nantong University 19 Qixiu Road Nantong;
Activation; Apoptosis; Hepatic fibrosis; Hepatic stellate cells; Schistosoma japonicum; Soluble egg antigen;
机译:日本血吸虫可溶性蛋抗原诱导凋亡并抑制肝星状细胞的激活:一种可能的分子机制
机译:通过STAT3 / P53 / P21途径激活Scistosoma的血吸虫血吸虫蛋瘤中的可溶性蛋诱导衰老
机译:血吸虫瘤可溶性蛋抗原抑制肝星状细胞中的TNF诱导的IL-34表达
机译:基质刚度与TGF-β1诱导的肝星状细胞活化的相关性
机译:胎球蛋白在活化的肝星状细胞中抑制TGF-β刺激的胶原蛋白合成并防止细胞凋亡。
机译:日本血吸虫的可溶性卵抗原通过激活FoxO3a / SKP2 / P27途径诱导活化的肝星状细胞衰老。
机译:日本血吸虫可溶性卵抗原通过激活FoxO3a / sKp2 / p27途径诱导活化肝星状细胞衰老。