机译:用线粒体复合物I在脑缺血/再灌注损伤中氧化还原依赖性损失Flavin
Columbia Univ Dept Pediat Div Neonatol William Black Bldg 650 W 168th St New York NY 10032 USA;
Columbia Univ Dept Pediat Div Neonatol William Black Bldg 650 W 168th St New York NY 10032 USA;
Columbia Univ Dept Pediat Div Neonatol William Black Bldg 650 W 168th St New York NY 10032 USA;
Weill Cornell Med Feil Family Brain &
Mind Res Inst New York NY USA;
Weill Cornell Med Feil Family Brain &
Mind Res Inst New York NY USA;
Weill Cornell Med Feil Family Brain &
Mind Res Inst New York NY USA;
Goethe Univ Med Sch Core Unit SFB815 Funct Prote Frankfurt Germany;
Columbia Univ Dept Pediat Div Neonatol William Black Bldg 650 W 168th St New York NY 10032 USA;
Columbia Univ Dept Pediat Div Neonatol William Black Bldg 650 W 168th St New York NY 10032 USA;
ischemia; reperfusion injury; mitochondrial complex I; flavin mononucleotide; reverse electron transfer; secondary energy failure;
机译:用线粒体复合物I在脑缺血/再灌注损伤中氧化还原依赖性损失Flavin
机译:黄素和谷胱甘肽在脑缺血/再灌注损伤中复合I介导的生物生物生物损伤的关键作用
机译:黄素和谷胱甘肽在脑缺血/再灌注损伤中复合I介导的生物生物生物损伤的关键作用
机译:MyRistoyaated肽(TAT)缀合的线粒体裂变肽抑制剂(P110)在心肌缺血/再灌注(I / R)损伤中的损伤效果比较
机译:心脏缺血再灌注损伤中的线粒体功能见解
机译:反向电子转移导致线粒体复合物的黄素丢失I:脑缺血再灌注损伤的潜在机制
机译:反向电子转移导致线粒体复合体I的黄素损失。脑缺血再灌注损伤的潜在机制