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机译:SOD3被脂肪细胞分泌并减轻高脂饮食诱导的肥胖,炎症和胰岛素抵抗
Center for Mitochondrial Biology and Medicine The Key Laboratory of Biomedical Information;
Department of Gastroenterology Xijing Hospital of Digestive Diseases State Key Laboratory of;
The Key Laboratory of Biomedical Information Engineering of the Ministry of Education School of;
The Key Laboratory of Biomedical Information Engineering of the Ministry of Education School of;
Center for Mitochondrial Biology and Medicine The Key Laboratory of Biomedical Information;
Department of Human Anatomy Histology and Embryology School of Basic Medical Sciences Xi'an;
Center for Mitochondrial Biology and Medicine The Key Laboratory of Biomedical Information;
Center for Mitochondrial Biology and Medicine The Key Laboratory of Biomedical Information;
Department of Human Anatomy Histology and Embryology School of Basic Medical Sciences Xi'an;
Department of Biochemistry and Molecular Biology School of Basic Medical Sciences Xi'an Jiaotong;
Faculty of Health and Medical Sciences University of Surrey;
Center for Mitochondrial Biology and Medicine The Key Laboratory of Biomedical Information;
SOD3; adipose tissue; lipid metabolism; metabolic pathways; obesity;
机译:SOD3被脂肪细胞分泌并减轻高脂饮食诱导的肥胖,炎症和胰岛素抵抗
机译:小鼠中诱导蛋白质信号调节-10的耗尽夸大了高脂饮食诱导的胰岛素抵抗和炎症,并且通过膳食绿茶提取物减轻了这种效果
机译:非雌激素的Xanthohumol衍生物减轻高脂饮食诱导的肥胖小鼠中的胰岛素抗性和认知障碍
机译:肥胖患者及与肥胖相关的胰岛素抵抗患者的FT-IR唾液分析
机译:解偶联蛋白3缺乏对高脂饮食诱导肥胖和胰岛素抵抗的影响
机译:咖啡摄入减轻了高脂饮食诱导的肥胖小鼠骨骼肌的炎症和肥胖诱导的胰岛素抵抗
机译:咖啡进气缓解炎症和肥胖诱导的高脂饮食诱导的肥胖小鼠骨骼肌中的胰岛素抗性