机译:尿激酶型纤溶酶原激活剂(UPA)对于实验性肾病综合征中的上皮钠通道(ENAC)介导的钠保留不是必需的
Department of Internal Medicine Division of Endocrinology Diabetology Vascular Disease;
Department of Internal Medicine Division of Endocrinology Diabetology Vascular Disease;
Department of Internal Medicine Division of Endocrinology Diabetology Vascular Disease;
Institute of Cellular and Molecular PhysiologyFriedrich‐Alexander University Erlangen‐Nürnberg (FAU;
Institute of Cellular and Molecular PhysiologyFriedrich‐Alexander University Erlangen‐Nürnberg (FAU;
Institute of Cellular and Molecular PhysiologyFriedrich‐Alexander University Erlangen‐Nürnberg (FAU;
Department of Internal Medicine Division of Endocrinology Diabetology Vascular Disease;
Department of Internal Medicine Division of Endocrinology Diabetology Vascular Disease;
Department of Internal Medicine Division of Endocrinology Diabetology Vascular Disease;
Department of Internal Medicine Division of Endocrinology Diabetology Vascular Disease;
Institute of Basic Medical SciencesUniversity of OsloOslo Norway;
Institute of AnatomyUniversity of RegensburgRegensburg Germany;
Institute of Cellular and Molecular PhysiologyFriedrich‐Alexander University Erlangen‐Nürnberg (FAU;
Department of Internal Medicine Division of Endocrinology Diabetology Vascular Disease;
Amiloride; epithelial sodium channel (ENaC); nephrotic syndrome; plasminogen; sodium retention; urokinase‐type plasminogen activator;
机译:尿激酶型纤溶酶原激活剂(UPA)对于实验性肾病综合征中的上皮钠通道(ENAC)介导的钠保留不是必需的
机译:肾病患者水肿和上皮钠通道激活尿纤溶酶原 - 纤溶酶的关系
机译:血浆kallikrein在体外激活上皮钠通道,但对肾病小鼠的体积保留不是必需的
机译:糖化胶原蛋白降低内皮细胞中尿激酶纤溶酶原激活物(UPA)的活性
机译:尿激酶和姜油调节人上皮钠通道(ENaC)。
机译:在鸡尿激酶型纤溶酶原激活物(ch-uPA)中引入RRHR基序可赋予对纤溶酶原激活物抑制剂(PAI)-1和PAI-2的敏感性并使ch-uPA介导的细胞外基质降解受PAI-1控制
机译:在鸡尿激酶型纤溶酶原激活物(ch-uPA)中引入RRHR基序可赋予对纤溶酶原激活物抑制剂(PAI)-1和PAI-2的敏感性,并使ch-uPA介导的细胞外基质降解受PAI-1控制