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机译:增加的下硫酸硫化硫化氢通过抑制PGE 2 2产生有助于内毒素耐受性
Department of PhysiologyUniversity of S?o PauloRibeir?o Preto Brazil;
Department of PhysiologyUniversity of S?o PauloRibeir?o Preto Brazil;
Department of PhysiologyUniversity of S?o PauloRibeir?o Preto Brazil;
Department of PhysiologyUniversity of S?o PauloRibeir?o Preto Brazil;
Department of PhysiologyUniversity of S?o PauloRibeir?o Preto Brazil;
Department of Basic and Oral BiologyUniversity of S?o PauloRibeir?o Preto Brazil;
corticosterone; cytokines; fever; gasotransmitters; inflammation; lipopolysaccharide;
机译:增加的下硫酸硫化硫化氢通过抑制PGE 2 2产生有助于内毒素耐受性
机译:硫化氢调节染色质重塑和炎症介质产生的响应内毒素,但在内毒素耐受性的发展中不起作用
机译:一氧化氮急性调节下丘脑和神经内肌肌型一氧化碳和硫化氢产生,以控制大鼠的血压加强素,催产素和心房利尿尿肽释放
机译:在北海高温/高盐度油田生产井中控制硫化氢清除剂诱导的水垢以及硫化物水垢形成的相关挑战。实验室评估到现场应用
机译:过量表达甘露醇-1-磷酸脱氢酶可增加甘露醇的产量,并赋予转基因矮牵牛cv耐盐和耐寒性。米切尔
机译:硫化氢调节内毒素响应的染色质重塑和炎症介质产生但在内毒素耐受性的发展中不起作用
机译:硫化氢调节内毒素响应的染色质重塑和炎症介质产生,但在内毒素耐受性的发展中不起作用