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机译:肝脏特异性ATG5 -Defige小鼠肝脏在肝脏特异性ATG5 -Defige液滴生物发生的受损通过持久的核因子样2激活介导
Department of Pharmacology Toxicology and Therapeutics The University of Kansas Medical Center;
Department of Pharmacology Toxicology and Therapeutics The University of Kansas Medical Center;
Institute of Chinese Materia Medica Shanghai University of Traditional Chinese Medicine;
Institute of Chinese Materia Medica Shanghai University of Traditional Chinese Medicine;
Department of Pharmacology Toxicology and Therapeutics The University of Kansas Medical Center;
Department of Pharmacology Toxicology and Therapeutics The University of Kansas Medical Center;
Department of Pharmacology Toxicology and Therapeutics The University of Kansas Medical Center;
机译:肝脏特异性ATG5 -Defige小鼠肝脏在肝脏特异性ATG5 -Defige液滴生物发生的受损通过持久的核因子样2激活介导
机译:肝特异性Atg5的丧失导致Nrf2的持续活化并保护对乙酰氨基酚引起的肝损伤
机译:Atg5的肝脏特异性丢失导致Nrf2的持久活化并防止对乙酰氨基酚引起的肝损伤。
机译:白介素6诱导的STAT3和AP-1放大肝细胞核因子1介导的肝基因反式激活,这是对肝损伤的适应性反应。
机译:持久性核因子样2激活介导的肝特异性Atg5缺陷型小鼠肝脏中空腹诱导的自适应脂质液滴生物发生受损。
机译:肝脏特异性Atg5缺乏小鼠肝脏中的禁食诱导的自适应脂质液滴生物发生在持续的核因子般的2激活中介导