机译:肿瘤引发干细胞出现自适应免疫抗性
Rockefeller Univ Howard Hughes Med Inst Robin Chemers Neustein Lab Mammalian Cell Biol &
New York NY 10065 USA;
Rockefeller Univ Howard Hughes Med Inst Robin Chemers Neustein Lab Mammalian Cell Biol &
New York NY 10065 USA;
Rockefeller Univ Howard Hughes Med Inst Robin Chemers Neustein Lab Mammalian Cell Biol &
New York NY 10065 USA;
Rockefeller Univ Howard Hughes Med Inst Robin Chemers Neustein Lab Mammalian Cell Biol &
New York NY 10065 USA;
Rockefeller Univ Howard Hughes Med Inst Robin Chemers Neustein Lab Mammalian Cell Biol &
New York NY 10065 USA;
Rockefeller Univ Howard Hughes Med Inst Robin Chemers Neustein Lab Mammalian Cell Biol &
New York NY 10065 USA;
Mem Sloan Kettering Canc Ctr Lab Epithelial Canc Biol Dept Surg New York NY 10065 USA;
Univ Calif San Francisco Dept Dermatol San Francisco CA 94143 USA;
Rockefeller Univ Howard Hughes Med Inst Robin Chemers Neustein Lab Mammalian Cell Biol &
New York NY 10065 USA;
机译:肿瘤引发干细胞出现自适应免疫抗性
机译:反义抑制肿瘤起始干样细胞中的microRNA-21和microRNA-221调节胰腺癌的肿瘤发生,转移和化疗耐药性
机译:肿瘤引发茎状细胞和耐药性:通过Toll样受体,环境因素和病毒来致癌作用
机译:内部专家的免疫学更新:了解先天和自适应免疫系统的细胞和分子
机译:微环境因素对套细胞淋巴瘤小鼠模型中潜在的肿瘤引发细胞的干细胞样和致瘤特性的影响。
机译:CD164促进具有干细胞活性的肺癌起始细胞并通过Akt / mTOR信号传导确定肿瘤的生长和耐药性
机译:肿瘤引发干细胞出现自适应免疫抗性