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机译:STAT3诱导的LNCRNA ABHD11-AS1通过调节miR-1301-3P / Stat3轴和PI3K / AKT信号通路来促进乳头状甲状腺癌中的肿瘤进展
Navy Gen Hosp Dept Radiat &
Oncol Beijing Peoples R China;
Navy Gen Hosp Dept Neurosurg Beijing Peoples R China;
Southern Med Univ TCM Integrated Canc Ctr Guangzhou Guangdong Peoples R China;
AnHui Med Univ Hefei Anhui Peoples R China;
Navy Gen Hosp Dept Radiat &
Oncol Beijing Peoples R China;
Navy Gen Hosp Dept Radiat &
Oncol Beijing Peoples R China;
Navy Gen Hosp Dept Radiat &
Oncol Beijing Peoples R China;
Navy Gen Hosp Dept Radiat &
Oncol Beijing Peoples R China;
Navy Gen Hosp Dept Radiat &
Oncol Beijing Peoples R China;
Navy Gen Hosp Dept Radiat &
Oncol Beijing Peoples R China;
lncRNA ABHD11-AS1; miR-1301-3p; papillary thyroid carcinoma; PI3K; AKT signalling pathway; STAT3;
机译:STAT3诱导的LNCRNA ABHD11-AS1通过调节miR-1301-3P / Stat3轴和PI3K / AKT信号通路来促进乳头状甲状腺癌中的肿瘤进展
机译:STAT3诱导的LNCRNA SNHG17上调预测黑素瘤的预后差,通过调节PI3K-AKT途径来促进细胞增殖和转移
机译:STAT3诱导的长非致RNA HNF1A-AS1的上调促进了通过激活Notch信号通路的口腔鳞状细胞癌的进展
机译:DLX4同源蛋白通过上调VEGFA促进ER阴性乳腺癌细胞中的PI3K / Akt抗凋亡途径。
机译:STAT3诱导的长时非编码RNA HNF1A-AS1的上调通过激活Notch信号通路促进口腔鳞状细胞癌的发展
机译:STAT3诱导的LNCRNA ABHD11-AS1通过调节miR-1301-3P / Stat3轴和PI3K / AKT信号通路来促进乳头状甲状腺癌中的肿瘤进展