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机译:SOD1联系肌营养侧刀状小鼠中胱抑素C的胱抑素C修复疾病
Department of Neuroscience and PathobiologyResearch Institute of Environmental MedicineChikusa‐ku;
Department of Neuroscience and PathobiologyResearch Institute of Environmental MedicineChikusa‐ku;
Department of Neuroscience and PathobiologyResearch Institute of Environmental MedicineChikusa‐ku;
Department of Life SciencesThe University of Tokyo KomabaMeguro‐ku Tokyo Japan;
Department of Neuroscience and PathobiologyResearch Institute of Environmental MedicineChikusa‐ku;
Amyotrophic lateral sclerosis ( ALS ); Cu/Zn superoxide dismutase ( SOD 1); Cystatin C (CysC); AMP ‐activated kinase ( AMPK );
机译:SOD1联系肌营养侧刀状小鼠中胱抑素C的胱抑素C修复疾病
机译:内源性调节性T淋巴细胞可减轻小鼠的肌萎缩性侧索硬化症,并与肌萎缩性侧索硬化症患者的疾病进展相关。
机译:内源性调节性T淋巴细胞可改善小鼠的肌萎缩性侧索硬化症,并与肌萎缩性侧索硬化症患者的疾病进展相关
机译:MARDI-TOF MS和FT-ICR MS的家族性肌萎缩外硬化相关交联Cu,Zn超氧化物歧化酶(SOD1)的分析
机译:CCS铜伴侣蛋白对与肌萎缩性侧索硬化症相关的SOD1突变体的影响
机译:脑室内给予胱抑素C改善SOD1连锁的肌萎缩性侧索硬化小鼠的疾病
机译:内源性调节性T淋巴细胞可减轻小鼠的肌萎缩性侧索硬化症,并与肌萎缩性侧索硬化症患者的疾病进展相关