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机译:TDP-43在神经变性中的病理机制
Departments of PathologyCase Western Reserve UniversityCleveland Ohio USA;
Departments of PathologyCase Western Reserve UniversityCleveland Ohio USA;
Departments of PathologyCase Western Reserve UniversityCleveland Ohio USA;
College of SciencesUniversity of Texas at San AntonioSan Antonio Texas USA;
Departments of PathologyCase Western Reserve UniversityCleveland Ohio USA;
Alzheimer's disease; amyotrophic lateral sclerosis; frontotemporal lobar degeneration; neurodegeneration; Neurodegenerative diseases; TDP ‐43;
机译:TDP-43在神经变性中的病理机制
机译:分子内盐桥链接TDP43 RNA结合,蛋白质稳定性和TDP43依赖神经变性。
机译:突变体TDP43患者衍生神经元的年龄依赖性神经变性和细胞器迁移缺陷与TDP43聚集无关
机译:早期检测到ALS患者组织工程皮肤中TDP-43的结构异常和细胞质积累
机译:小伸长率复合物(LEC)和超伸长率复合物(SEC)作为TDP-43相关神经变性的调节剂
机译:TDP-43在神经退行性病变中的机制
机译:Trem2与TDP-43相互作用,并介导针对与TDP-43相关神经变性的微胶质神经保护作用