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Rational Roots of Sympathetic Overactivity by Neurogenic Pulmonary Edema Modeling Arising from Sympathyco-Vagal Imbalance in Subarachnoid Hemorrhage: An Experimental Study

机译:来自蛛网膜下腔出血中同情肺水肿模拟的神经肺水肿建模的理性根源:实验研究

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摘要

BACKGROUND: Autonomous innervations of the lungs are maintained by cervical sympathetic and vagal nerves. Sympathetic overactivity-induced neurogenic pulmonary edema (NPE) is known as a serious complication of subarachnoid hemorrhage, but the rational neuronal mechanism of that overactivity has not yet been clarified fully. The aim of this study was to examine whether there is a relationship between vagal nerve ischemia related sympathetic overactivity and neurogenic pulmonary edema in subarachnoid hemorrhage.
机译:背景:肺部的自主支护由宫颈交感神经和迷走神经保持。 交感神经过度诱导的神经源性肺水肿(NPE)被称为蛛网膜下腔出血的严重并发症,但尚未完全澄清该过度率的理性神经元机制。 本研究的目的是审查蛛网膜下腔中的迷血神经缺血相关交感神经过度活动和神经源性肺水肿之间是否存在关系。

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