机译:Trim32,但不是其肌肉营养不良相关的突变体,正面调节并靶向P62 / SQSTM1的自噬降解
Arctic Univ Norway Univ Tromso Dept Med Biol Mol Canc Res Grp N-9037 Tromso Norway;
Arctic Univ Norway Univ Tromso Dept Med Biol Mol Canc Res Grp N-9037 Tromso Norway;
Arctic Univ Norway Univ Tromso Dept Med Biol Mol Canc Res Grp N-9037 Tromso Norway;
Arctic Univ Norway Univ Tromso Dept Med Biol Mol Canc Res Grp N-9037 Tromso Norway;
Arctic Univ Norway Univ Tromso Dept Med Biol Mol Canc Res Grp N-9037 Tromso Norway;
Arctic Univ Norway Univ Tromso Dept Med Biol Mol Canc Res Grp N-9037 Tromso Norway;
Univ New Mexico Hlth Sci Ctr Autophagy Inflammat &
Metab Ctr Biomed Res Excell Albuquerque NM;
Arctic Univ Norway Univ Tromso Dept Med Biol Mol Canc Res Grp N-9037 Tromso Norway;
Arctic Univ Norway Univ Tromso Dept Med Biol Mol Canc Res Grp N-9037 Tromso Norway;
Arctic Univ Norway Univ Tromso Dept Med Biol Mol Canc Res Grp N-9037 Tromso Norway;
TRIM32; p62/SQSTM1; LGMD2H; Autophagy; BBS11; Ubiquitylation;
机译:Trim32,但不是其肌肉营养不良相关的突变体,正面调节并靶向P62 / SQSTM1的自噬降解
机译:两种LC3结合蛋白SQSTM1 / p62和TBC1D25 / OATL1对自噬降解的敏感性不同
机译:蛋白毒性应激通过ULK1诱导p62 / SQSTM1的磷酸化,以调节蛋白质聚集体的选择性自噬清除。
机译:poly c结合蛋白1调节p62 / sqstm1 mRNA稳定性和自噬降解以抑制肿瘤进展
机译:Trim32作为基质和肌营养不良症患者的P62 / SQSTM1的阳性调节剂作用