机译:颅内自刺激和固定化对PC12M3细胞中的神经突延伸和P38 MAPK途径产生了不同的影响
Department of Pharmacy Okayama University Hospital;
Department of Pharmacy Okayama University Hospital;
Department of Pharmacy Okayama University Hospital;
Graduate School of Health of Science Kibi International University;
Department of Pharmacy Okayama University Hospital;
Center for Clinical Pharmacy Matsuyama University;
Division of Pharmacy Ehime University Hospital;
School of Pharmacy Shujitsu University;
Graduate School of Health of Science Kibi International University;
Graduate School of Health of Science Kibi International University;
Intracranial self-stimulation-reward; Immobilization-aversion; PC12 variant cells; p38 MAPK pathway;
机译:颅内自刺激和固定化对PC12M3细胞中的神经突延伸和P38 MAPK途径产生了不同的影响
机译:源自蜂胶的青霉素C通过ERK和p38 MAPK途径诱导PC12m3细胞中神经突生长。
机译:源自蜂胶的青霉素C通过ERK和p38 MAPK途径在PC12m3细胞中诱导神经突生长。
机译:开发p38 MAPK和糖皮质激素信号通路的新型整合模型
机译:MAPK级联p38,JNK和ERK在调节人神经元冷却应激模型中神经突向外生长中的作用
机译:通过p38丝裂原激活的蛋白激酶途径对比PC12m3细胞中浴诱导的神经突生长
机译:p38MAPK磷酸化paxillin参与PC-12细胞的神经突延伸