机译:Colivelin拯救缺血神经元和伴随Jak / Stat3信号通路的轴突
Shanxi Med Univ Taiyuan Shanxi Peoples R China;
Tianjin Med Univ Tianjin Neurol Inst Dept Neurol Gen Hosp Tianjin Peoples R China;
Tianjin Med Univ Tianjin Neurol Inst Dept Neurol Gen Hosp Tianjin Peoples R China;
Tianjin Med Univ Tianjin Neurol Inst Dept Neurol Gen Hosp Tianjin Peoples R China;
Third Peoples Hosp Datong Ctr Neurol Dis Datong Shanxi Peoples R China;
Third Peoples Hosp Datong Ctr Neurol Dis Datong Shanxi Peoples R China;
Third Peoples Hosp Datong Ctr Neurol Dis Datong Shanxi Peoples R China;
Capital Med Univ Beijing Tiantan Hosp China Natl Clin Res Ctr Neurol Dis Beijing Peoples R China;
Shanxi Med Univ Taiyuan Shanxi Peoples R China;
ischemic stroke; colivelin; neuronal death; axonal remodeling; JAK/STAT3;
机译:Colivelin拯救缺血神经元和伴随Jak / Stat3信号通路的轴突
机译:SOCS1-JAK2-STAT3信号通路的抑制赋予缺血性中风大鼠神经保护。
机译:亚洲酸改善CC 1 4 sub>诱导的大鼠肝纤维化:Nrf2 / ARE,NF-κB/IκBα和JAK1 / STAT3信号通路的参与
机译:Abeta通过在海马神经元中扰乱JAK2 / Stat3轴来引起内存损伤。
机译:过度活跃的Alpha2-Chimaerin揭示了运动神经元发育中轴突指导信号通路的复杂性。
机译:棕榈酰酰基转移酶ZDHHC5和ZDHHC8在DRG轴突中唯一存在并通过GP130 / JAK / Stat3路径控制逆行信号传导
机译:生脉散三种成分的组合通过基于生物信息学方法调节mapKs和JaK2-sTaT3信号通路保护心肌缺血性疾病并抑制氧化应激