机译:氮化体系涉及氮蛋白诱导地位癫痫毒性抗抑郁作用的影响:INOS和COX-2基因表达的评价
Tehran University of Medical Sciences Faculty of Pharmacy Pharmacology and Toxicology;
Tehran University of Medical Sciences Faculty of Pharmacy Pharmacology and Toxicology;
Experimental Medicine Research Center Tehran University of Medical Sciences;
Hematology/Oncology and Stem Cell Transplantation Research Center Shariati Hospital School of;
Tehran University of Medical Sciences Faculty of Pharmacy Pharmacology and Toxicology;
Tehran University of Medical Sciences Faculty of Pharmacy Pharmacology and Toxicology;
Experimental Medicine Research Center Tehran University of Medical Sciences;
Status epilepticus; Zolpidem; Nitric oxide; Cyclooxygenase; Inducible nitric oxide synthase;
机译:氮化体系涉及氮蛋白诱导地位癫痫毒性抗抑郁作用的影响:INOS和COX-2基因表达的评价
机译:早期的Endocannabinoid系统激活衰减初始影响诱导的行为损伤,但不能预防锂毒性野兔状态癫痫模型中的癫痫发作
机译:锂-毛果芸香碱诱发癫痫持续状态(PISE)后,海马CA1区Na + -K + -Cl-协同转运蛋白1(NKCC1)和K + -Cl-协同转运蛋白2(KCC2)的长期表达变化。
机译:通过InOS和COX-2表达的改变,通过关节软骨细胞的动态压缩影响白细胞介素-1beta诱导的一氧化氮和前列腺素E_2释放
机译:通过C端β-Arrestin磷酸传感器对游离脂肪酸受体4(FFA4)对ROS生成和COX-2表达的调节的评估
机译:锂-毛果芸香碱诱发癫痫持续状态后未成熟和成年大鼠海马中活化转录因子2和c-Jun的差异表达
机译:锂-毛果芸香碱诱发癫痫持续状态后未成熟和成年大鼠海马中活化转录因子2和c-Jun的差异表达
机译:soman诱导的大鼠癫痫持续状态后白细胞介素-18表达对神经血管损伤的反应增加。