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首页> 外文期刊>Journal of Electrocardiology: An International Publication for the Study of the Electrical Activities of the Heart >Longitudinal dissociation and concealed electrotonic conduction as a possible mechanism of AV block.
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Longitudinal dissociation and concealed electrotonic conduction as a possible mechanism of AV block.

机译:纵向解离和隐藏的电渗传导是房室传导阻滞的可能机制。

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摘要

We have read with much interest a recent report by Bhargava et al on AV block. They represented an electrocardiogram showing sinus tachycardia with sinus rate exceeding the ventricular rate suggesting atrioventricular (AV) block in a patient with old anterior wall infarction. Fig. 1 is the electrocardiogram shown in their report, in which the presence of varying PR intervals, irregular R-R intervals and P-QRS relationship not consistent with second-degree, type I AV block was seen. As represented in the upper diagram of Fig. 1, they explained that 2-level block occurred in the AV junction as the mechanism.In the electrocardiogram, sinus impulse SI is conducted to the ventricles with a normal PR interval. The next sinus impulse, S2, is conducted with a markedly long PR interval of 0.68 second. In cases of our previous report, such abrupt marked prolongation of the PR interval was explained by longitudinal dissociation in the AV junction. In this letter, attempts are made to explain the case not by 2-level AV block but by longitudinal dissociation and concealed electrotonic conduction in the AV junction as an alternative possible mechanism, as shown in the lower diagram of Fig. 1.
机译:我们非常感兴趣地阅读了Bhargava等人有关房室传导阻滞的最新报告。他们代表了一个心电图,显示窦性心动过速,窦率超过心室率,提示患有旧前壁梗死的患者房室(AV)阻滞。图1是他们的报告中显示的心电图,其中发现存在变化的PR间隔,不规则的R-R间隔和P-QRS关系,与二度I型AV阻滞不一致。如图1的上图所示,他们解释了在AV连接处发生2级阻塞的机制。在心电图中,窦性冲动SI以正常的PR间隔传导至心室。下一个窦性脉冲S2的PR间隔很长,为0.68秒。在我们先前的报告中,PR间期的这种明显延长是由AV结的纵向分离所解释的。在这封信中,试图通过2级AV块而不是2级AV块来解释这种情况,如图1的下部图所示,它是通过在AV结中进行纵向解离和隐藏的电渗传导作为一种可能的替代机制。

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