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Isotropic continuum damage/repair model for alveolar bone remodeling

机译:各向同性连续体损伤/修复模型用于牙槽骨重塑

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摘要

Several authors have proposed mechanical models to predict long term tooth movement, considering both the tooth and its surrounding bone tissue as isotropic linear elastic materials coupled to either an adaptative elasticity behavior or an update of the elasticity constants with density evolution. However, tooth movements obtained through orthodontic appliances result from a complex biochemical process of bone structure and density adaptation to its mechanical environment, called bone remodeling. This process is far from linear reversible elasticity. It leads to permanent deformations due to biochemical actions. The proposed biomechanical constitutive law, inspired from Doblar and Garca (2002) [30], is based on a elasto-viscoplastic material coupled with Continuum isotropic Damage Mechanics (Doblar and Garca (2002) [30] considered only the case of a linear elastic material coupled with damage). The considered damage variable is not actual damage of the tissue but a measure of bone density. The damage evolution law therefore implies a density evolution. It is here formulated as to be used explicitly for alveolar bone, whose remodeling cells are considered to be triggered by the pressure state applied to the bone matrix. A 2D model of a tooth submitted to a tipping movement, is presented. Results show a reliable qualitative prediction of bone density variation around a tooth submitted to orthodontic forces.
机译:几位作者提出了机械模型来预测牙齿的长期运动,将牙齿及其周围的骨组织都视为各向同性的线性弹性材料,这些材料与适应性的弹性行为或随着密度变化的弹性常数的更新耦合在一起。但是,通过正畸矫治器获得的牙齿运动是由复杂的骨骼结构生化过程及其对机械环境的密度适应(称为骨骼重塑)引起的。这个过程远非线性可逆弹性。由于生化作用,它会导致永久变形。受Doblar和Garca(2002)[30]的启发,提出的生物力学本构律是基于弹性粘塑性材料和连续各向同性损伤力学(Doblar和Garca(2002)[30]),仅考虑了线性弹性的情况。材料加上损坏)。所考虑的损伤变量不是组织的实际损伤,而是骨密度的量度。因此,损伤演化定律意味着密度演化。它在这里被配制为明确用于牙槽骨,其重塑细胞被认为是由施加到骨基质上的压力状态触发的。呈现了进行倾翻运动的牙齿的2D模型。结果表明,在正畸力作用下,牙齿周围骨密度变化的可靠定性预测。

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