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机译:RAX / PKR协会中不足的PKR改善了乙醇诱导的小脑发育中的神经毒性。
Department Pharmacology Nutritional Sciences University of Kentucky College of Medicine">(1);
The First Affiliated Hospital of Xiamen University">(2);
Department of Biochemistry and Molecular Biology School of Basic Medical Sciences Fujian Medical University">(3);
Graduate Center for Toxicology University of Kentucky College of Medicine">(4);
Department of Physiology and Pathophysiology School of Basic Medical Sciences Fujian Medical University">(5);
Department Pharmacology Nutritional Sciences University of Kentucky College of Medicine">(1);
University of Kentucky">(6);
Department Pharmacology Nutritional Sciences University of Kentucky College of Medicine">(1);
Department of Neurology Affiliated Provincial Hospital of Anhui Medical University">(7);
Department Pharmacology Nutritional Sciences University of Kentucky College of Medicine">(1);
Alcohol; Neuron; Cerebellum; Apoptosis; Fetal alcohol spectrum disorders; PKR; Rax;
机译:MicroRNA-29B通过SP1 / Rax / PKR级联调节乙醇诱导的乙醇诱导的神经元细胞凋亡
机译:原发性素受体多态性之间的关系(PKR1 RS4627609和PKR2 RS6053283)和复发性妊娠损失
机译:RAX / PACT-PKR应激反应途径促进p53的磺酰化和激活,导致G 1阻滞
机译:在发育中的大鼠小脑中,氧化应激和胱天蛋白酶激活在乙醇诱导的细胞损失中的作用。
机译:RAX / PKR协会中不足的PKR改善了乙醇诱导的小脑发育中的神经毒性。
机译:MicroRNA-29B通过SP1 / Rax / PKR级联调节乙醇诱导的乙醇诱导的神经元细胞凋亡