机译:轴突静脉曲张中的ERK激活通过突触素I调节海马CA3区的突触前可塑性
Department of Anatomy, Pharmacology, and Forensic Medicine, University of Turin, C.so Massimo d'Azeglio, 52, I-10126 Turin, Italy National Institute of Neuroscience, 10125 Turin, Italy;
National Institute of Neuroscience, 10125 Turin, Italy Department of Neuroscience and Brain Technologies, The Italian Institute of Technology, Via Morego 30, I-16131 Genova, Italy Department of Experimental Medicine, University of Genova, 16126 Genova, Italy;
National Institute of Neuroscience, 10125 Turin, Italy Department of Neuroscience and Brain Technologies, The Italian Institute of Technology, Via Morego 30, I-16131 Genova, Italy Department of Experimental Medicine, University of Genova, 16126 Genova, Italy;
Department of Anatomy, Pharmacology, and Forensic Medicine, University of Turin, C.so Massimo d'Azeglio, 52, I-10126 Turin, Italy National Institute of Neuroscience, 10125 Turin, Italy;
Department of Anatomy, Pharmacology, and Forensic Medicine, University of Turin, C.so Massimo d'Azeglio, 52, I-10126 Turin, Italy National Institute of Neuroscience, 10125 Turin, Italy;
MAP kinase; mossy fibers; post-tetanic potentiation;
机译:NMDA和AMPA /海藻酸谷氨酸受体调节正常和缺血性海马中的齿状神经发生和CA3突触蛋白-I。
机译:PDE2A的抑制作用,而非PDE9A的抑制作用,通过海马CA1区的成对脉冲促进作用来调节突触前的短期可塑性。
机译:Bourne,J.N.a c,Chirillo,M.a. a b,Harris,K.M.a在成熟海马的长期增强过程中沿CA3→CA1轴突的突触前超结构可塑性
机译:海马CA3区反复侧支连续时间递归神经网络模型
机译:输入到CA3中间神经的生苔纤维:平衡短期可塑性和突触前受体的调节。
机译:轴突静脉曲张中的ERK激活通过突触素I调节海马CA3区的突触前可塑性
机译:轴突静脉曲张中的ERK激活通过突触素I调节海马CA3区的突触前可塑性