机译:JNK的遗传和药理抑制作用通过干扰VEGF的表达来改善缺氧性视网膜病变
Laboratory of Gene Regulation and Signal Transduction, University of California at San Diego, 9500 Gilman Drive, La Jolla, CA 92093-0723 Departments of Pharmacology, University of California at San Diego, 9500 Gilman Drive, La Jolla, CA 92093-0723 Pathology, University of California at San Diego, 9500 Gilman Drive, La Jolla, CA 92093-0723;
Laboratory of Gene Regulation and Signal Transduction, University of California at San Diego, 9500 Gilman Drive, La Jolla, CA 92093-0723 Departments of Pharmacology, University of California at San Diego, 9500 Gilman Drive, La Jolla, CA 92093-0723 Pathology, University of California at San Diego, 9500 Gilman Drive, La Jolla, CA 92093-0723;
Hamilton Glaucoma Center, University of California at San Diego, 9500 Gilman Drive, La Jolla, CA 92093-0723;
Pediatrics, School of Medicine, University of California at San Diego, 9500 Gilman Drive, La Jolla, CA 92093-0723;
Hamilton Glaucoma Center, University of California at San Diego, 9500 Gilman Drive, La Jolla, CA 92093-0723;
Laboratory of Gene Regulation and Signal Transduction, University of California at San Diego, 9500 Gilman Drive, La Jolla, CA 92093-0723 Departments of Pharmacology, University of California at San Diego, 9500 Gilman Drive, La Jolla, CA 92093-0723 Pathology, University of California at San Diego, 9500 Gilman Drive, La Jolla, CA 92093-0723;
neoangiogenesis; retinopathy of prematurity;
机译:通过靶向PI3K / AKT途径抑制HIF-1α/ VEGF表达,Gambogic酸改善糖尿病诱导的增殖性视网膜病变
机译:四甲基吡嗪通过HIF-1?/ JNK / p38和IGFBP3 / BNIP3抑制作用改善缺氧诱导的心肌细胞凋亡,从而上调PI3K / Akt生存信号。
机译:转录因子调节VPR91介导的VEGF在缺氧诱导的视网膜病变中的表达
机译:基于透明质酸的多价VEGF拮抗剂抑制糖尿病性视网膜病变中的致病性血管生成。
机译:小鼠尼古丁代谢研究:药理抑制作用和遗传影响对尼古丁药理的影响。
机译:JNK的遗传和药理抑制作用通过干扰VEGF的表达来改善缺氧性视网膜病变
机译:JNK的遗传和药理抑制作用通过干扰VEGF的表达来改善缺氧性视网膜病变