Tetramethylpyrazine Ameliorated Hypoxia-Induced Myocardial Cell Apoptosis via HIF-1?±/JNK/p38 and IGFBP3/BNIP3 Inhibition to Upregulate PI3K/Akt Survival Signaling

Kuan-Ho Lin; Wei-Wen Kuo; Ai-Zhi Jiang; Peiying Pai; Jing-Ying Lin; Wei-Kung Chen; Cecilia Hsuan Day; Chia-Yao Shen; V. Vijaya Padma; Chih Yang Huang

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Tetramethylpyrazine Ameliorated Hypoxia-Induced Myocardial Cell Apoptosis via HIF-1?±/JNK/p38 and IGFBP3/BNIP3 Inhibition to Upregulate PI3K/Akt Survival Signaling

机译：四甲基吡嗪通过HIF-1？/ JNK / p38和IGFBP3 / BNIP3抑制作用改善缺氧诱导的心肌细胞凋亡，从而上调PI3K / Akt生存信号。

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来源
《Cellular Physiology and Biochemistry 》 |2015年第1期| 共11页
作者
Kuan-Ho Lin; Wei-Wen Kuo; Ai-Zhi Jiang; Peiying Pai; Jing-Ying Lin; Wei-Kung Chen; Cecilia Hsuan Day; Chia-Yao Shen; V. Vijaya Padma; Chih Yang Huang;
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中图分类细胞生物物理学 ;
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1. Proteasome inhibitor MG132 induces selective apoptosis in glioblastoma cells through inhibition of PI3K/Akt and NFkappaB pathways, mitochondrial dysfunction, and activation of p38-JNK1/2 signaling [J] . Alfeu Zanotto-Filho, Elizandra Braganhol, Ana Maria Oliveira Battastini, Investigational New Drugs . 2012 ,第6期

机译：蛋白酶体抑制剂MG132通过抑制PI3K / Akt和NFkappaB途径，线粒体功能障碍以及激活p38-JNK1 / 2信号传导，诱导胶质母细胞瘤细胞选择性凋亡。
2. Proteasome inhibitor MG132 induces selective apoptosis in glioblastoma cells through inhibition of PI3K/Akt and NFkappaB pathways, mitochondrial dysfunction, and activation of p38-JNK1/2 signaling. [J] . Alfeu Zanotto-Filho, Elizandra Braganhol, Ana Maria Oliveira Battastini, Investigational new drugs. . 2012 ,第6期

机译：蛋白酶体抑制剂MG132通过抑制PI3K / Akt和NFkappaB途径，线粒体功能障碍以及激活p38-JNK1 / 2信号传导来诱导胶质母细胞瘤细胞选择性凋亡。
3. Isoorientin Induces Apoptosis and Autophagy Simultaneously by Reactive Oxygen Species (ROS)-Related p53, PI3K/Akt, JNK, and p38 Signaling Pathways in HepG2 Cancer Cells [J] . Li Yuan, Shuping Wei, Jing Wang Journal of Agricultural and Food Chemistry . 2014 ,第23期

机译：异Orientinin诱导HepG2癌细胞中活性氧（ROS）相关的p53，PI3K / Akt，JNK和p38信号通路同时诱导凋亡和自噬
4. Caspase Activation and Extracellular Signal-Regulated Kinase/Akt Inhibition Were Involved in Luteolin-Induced Apoptosis in Lewis Lung Carcinoma Cells [C] . JIN-HYUNG KIM, EUN-OK LEE, HYO-JUNG LEE, Meeting on Cell Signaling World: Signal Transduction Pathways as Therapeutic Targets . 2007

机译：半胱氨酸天冬氨酸蛋白酶激活和细胞外信号调节激酶/ Akt抑制涉及路易斯-肺癌细胞中木犀草素诱导的细胞凋亡。
5. The multi-targeted receptor tyrosine kinase inhibitor, linifanib (ABT-869), induces apoptosis and inhibits proliferation of leukemia cells through phosphoinositide-3 kinase (PI3K)/AKT-dependent signaling pathways. [D] . Hernandez, Jenny Elizabeth. 2010

机译：多靶点受体酪氨酸激酶抑制剂利尼法尼（ABT-869）通过磷酸肌醇3激酶（PI3K）/ AKT依赖性信号传导途径诱导凋亡并抑制白血病细胞的增殖。
6. Suppression of p38 MAPK and JNK via Akt-mediated Inhibition of Apoptosis Signal-regulating Kinase 1 Constitutes a Core Component of the β-Cell Pro-survival Effects of Glucose-dependent Insulinotropic Polypeptide [O] . Scott B. Widenmaier, Ziliang Ao, Su-Jin Kim, 2009

机译：通过Akt介导的凋亡信号调节激酶1抑制抑制p38 MAPK和JNK构成了葡萄糖依赖性促胰岛素多肽的β细胞促生存作用的核心成分。
7. Tetramethylpyrazine Ameliorated Hypoxia-Induced Myocardial Cell Apoptosis via HIF-1α/JNK/p38 and IGFBP3/BNIP3 Inhibition to Upregulate PI3K/Akt Survival Signaling [O] . Kuan-Ho Lin, Wei-Wen Kuo, Ai-Zhi Jiang, 2015

机译：川芎嗪通过HIF-1α/ JNK / p38和IGFBp3 / BNIp3抑制pI3K / akt生存信号上调改善缺氧诱导的心肌细胞凋亡

Tetramethylpyrazine Ameliorated Hypoxia-Induced Myocardial Cell Apoptosis via HIF-1?±/JNK/p38 and IGFBP3/BNIP3 Inhibition to Upregulate PI3K/Akt Survival Signaling

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