机译:内源性抗体促进神经损伤后快速的髓鞘清除和有效的轴突再生
Departments of Neurobiology, Stanford University School of Medicine, Stanford, CA 94305-5125;
rnDepartments of Neurobiology, Stanford University School of Medicine, Stanford, CA 94305-5125;
rnDepartments of Neurobiology, Stanford University School of Medicine, Stanford, CA 94305-5125;
rnDepartments of Immunology and Rheumatology, Stanford University School of Medicine, Stanford, CA 94305-5125;
rnDepartments of Neurobiology, Stanford University School of Medicine, Stanford, CA 94305-5125;
humoral immunity; nerve regeneration;
机译:SIRPα(信号调节蛋白-α)的缺失促进了Wallerian变性,轴突再生和神经损伤恢复中髓磷脂碎片的吞噬清除。
机译:神经动员促进神经生长因子促进神经再生,坐骨神经损伤后髓磷脂蛋白零增加
机译:锌螯合和KLF9敲低合作促进视神经损伤后轴突再生
机译:Neuriite-Myelin在Purkinje Axon生长和再生控制中的相互作用
机译:神经损伤后的长期基因表达:轴突和体细胞mRNA有助于海螺神经元的轴突再生。
机译:内源性抗体促进神经损伤后快速的髓鞘清除和有效的轴突再生
机译:内源性抗体促进神经损伤后快速的髓鞘清除和有效的轴突再生