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机译:C型凝集素样受体NKp65和KACL的相互作用促进了人类角质形成细胞的专门免疫识别
Department of Immunology, Institute for Cell Biology, Eberhard-Karls-University Tuebingen, 72076 Tuebingen, Germany;
rnDepartment of Immunology, Institute for Cell Biology, Eberhard-Karls-University Tuebingen, 72076 Tuebingen, Germany;
rnDepartment of Immunology, Institute for Cell Biology, Eberhard-Karls-University Tuebingen, 72076 Tuebingen, Germany;
rnDepartment of Immunology, Institute for Cell Biology, Eberhard-Karls-University Tuebingen, 72076 Tuebingen, Germany;
rnDepartment of Dermatology, Eberhard-Karls-University Tuebingen, 72076 Tuebingen, Germany;
rnDepartment of Immunology, Institute for Cell Biology, Eberhard-Karls-University Tuebingen, 72076 Tuebingen, Germany Institute for Molecular Medicine, Goethe-University Frankfurt, 60590 Frankfurt, Germany;
CLEC2 family; natural killer cells; NKRP1 receptors; skin;
机译:KACL的膜远端表面上的关键残基决定了角质形成细胞特异性C型凝集素样受体KACL及其高亲和力受体NKp65的功能相互作用,而不是糖基化
机译:激活的C型凝集素样受体NKp65信号通过基于半免疫受体酪氨酸的活化基序(hemITAM)和脾酪氨酸激酶(Syk)
机译:激活的C型凝集素样受体NKp65信号通过基于半免疫受体酪氨酸的活化基序(hemITAM)和脾酪氨酸激酶(Syk)
机译:凝集素样氧化低密度脂蛋白受体-1(LOX-1)在体外流量下对人脑微血管内皮细胞的腺苷二磷酸活性血小板粘附的作用
机译:RNAi抑制p53表达可诱导EGF受体水平,并促进人角质形成细胞中HPV16 E7永生化。
机译:C型凝集素样受体NKp65和KACL的相互作用促进了人类角质形成细胞的专门免疫识别
机译:C型凝集素样受体NKp65和KACL的相互作用促进了人类角质形成细胞的专门免疫识别
机译:登革病毒感染贴壁人单核细胞的两种机制的证据:胰蛋白酶敏感性病毒受体和胰蛋白酶抗性免疫复合物受体