机译:补体因子I控制的结构基础及其与疾病相关的序列多态性
Sir William Dunn School of Pathology, University of Oxford, Oxford 0X1 3RE, United Kingdom;
Sir William Dunn School of Pathology, University of Oxford, Oxford 0X1 3RE, United Kingdom;
Sir William Dunn School of Pathology, University of Oxford, Oxford 0X1 3RE, United Kingdom;
Sir William Dunn School of Pathology, University of Oxford, Oxford 0X1 3RE, United Kingdom;
Sir William Dunn School of Pathology, University of Oxford, Oxford 0X1 3RE, United Kingdom;
Department of Biochemistry, University of Oxford, Oxford 0X1 3QU, United Kingdom;
Department of Infection, Immunity and Biochemistry, School of Medicine, Cardiff University, Cardiff CF14 4XN, Wales,United Kingdom;
Department of Infection, Immunity and Biochemistry, School of Medicine, Cardiff University, Cardiff CF14 4XN, Wales,United Kingdom;
Department of Biochemistry, University of Oxford, Oxford 0X1 3QU, United Kingdom;
Sir William Dunn School of Pathology, University of Oxford, Oxford 0X1 3RE, United Kingdom;
allostery; innate immunity; atypical hemolytic-uremic syndrome; serine protease;
机译:使用序列特异性引物PCR和限制性片段长度多态性确定补体因子H功能多态性(V62I,Y402H和E936D)。
机译:HindIII限制性片段长度多态性分析揭示了人类补体C4A无效等位基因(C4A * Q0)的结构基础上的异质性
机译:H因子与疾病相关的序列变异:一种结构生物学方法
机译:靶向靶向疾病相关多态性的收敛EQTL分子和途径的精确药物重新估算
机译:肺炎球菌粘附和补体逃逸的分子基础:肺炎球菌毒力因子CbpA的结构和生化研究。
机译:补体因子I控制的结构基础及其与疾病相关的序列多态性
机译:补体因子I控制的结构基础及其与疾病相关的序列多态性