机译:含病毒性卵巢肿瘤结构域蛋白酶去除泛素和干扰素刺激基因15的结构基础
Department of Microbiology, University of Manitoba, Winnipeg, MB, R3T 2N2 Canada;
Laboratory of Virology and Infectious Disease, The Rockefeller University,1230 York Avenue, Box 64, New York, NY 10065;
Department of Microbiology, University of Manitoba, Winnipeg, MB, R3T 2N2 Canada;
Department of Microbiology Mount Sinai School of Medicine, New York, NY 10029;
Department of Chemistry, University of Manitoba, Winnipeg, MB, R3T 2N2 Canada;
Department of Microbiology Mount Sinai School of Medicine, New York, NY 10029,Department of Medicine, Division of Infectious Diseases Mount Sinai School of Medicine, New York, NY 10029,Global Health and Emerging Pathogens Institute,Mount Sinai School of Medicine, New York, NY 10029;
Department of Microbiology, University of Manitoba, Winnipeg, MB, R3T 2N2 Canada;
innate immunity; viral deubiquitinase; bunyavirus; crimean-congo haemorrhagic fever virus; viral immune evasion;
机译:确定特异性特异性干扰素刺激基因产物的分子驱动器15与奈病毒卵巢肿瘤域蛋白酶的相互作用
机译:病毒性卵巢肿瘤域中泛素和ISG15交叉反应性的分子基础
机译:泛素特异性蛋白酶18的分子表征揭示了干扰素刺激基因15的底物特异性
机译:卵巢肿瘤的组合非线性激光成像(双光子激发荧光,二次谐波和三次谐波生成以及荧光寿命成像显微镜检查)
机译:ISG15蛋白偶联物具有病毒免疫逃避蛋白酶靶向的抗病毒功能
机译:含病毒性卵巢肿瘤结构域蛋白酶去除泛素和干扰素刺激基因15的结构基础
机译:含病毒性卵巢肿瘤结构域蛋白酶去除泛素和干扰素刺激基因15的结构基础
机译:卵巢浆液性癌的发病机制是免疫定向诊断和治疗的基础。项目1 - 沿不同途径发展的卵巢浆液性肿瘤的分子特征