机译:泛素变体ubR48的表达降低拟南芥中的蛋白水解活性并诱导细胞死亡
Institute of Botany University of ViennaDepartment of Chromosome Biology Vienna Biocenter 2 University of Vienna;
Department of Plant Developmental Biology Max Planck Institute for Plant Breeding Research;
Institute of Botany University of ViennaDepartment of Chromosome Biology Vienna Biocenter 2 University of Vienna;
Institute of Botany University of ViennaCenter for Brain Research Medical University of Vienna;
Institute of Botany University of ViennaDepartment of Plant Developmental Biology Max Planck Institute for Plant Breeding Research;
Cell death; Pathogen response; Proteolysis inhibition; Reactive oxygen intermediates; Salicylic acid; Senescence; Ubiquitin–proteasome pathway;
机译:泛素变体ubR48的表达降低拟南芥中的蛋白水解活性并诱导细胞死亡
机译:泛素-蛋白酶体蛋白水解途径抑制剂对发育中的视网膜诱导的细胞死亡的分化依赖性敏感性。
机译:缺少其激活域的Ets1转录因子的表达降低了uPA的蛋白水解活性和细胞运动性,并损害了乳腺上皮细胞中正常的肾小管生成和癌性扩散
机译:LPS通过降低PPARγ表达和活性,诱导甘油三酯合成和分泌物的减少
机译:阿片类物质和IL -4对炎性和T辅助细胞反应的调节:对细胞因子产生,死亡效应子表达和激活诱导的细胞死亡的直接影响
机译:拟南芥植物U-BOX17的E3泛素连接酶活性及其功能性烟草同源物ACRE276是细胞死亡和防御所必需的
机译:遍在蛋白变体ubR48的表达降低拟南芥中的蛋白水解活性并诱导细胞死亡