机译:BTB-ZF因子募集E3连接酶cullin 3来调节淋巴样效应子程序
Committee on Immunology, Department of Pathology, The Howard Hughes Medical Institute, University of Chicago, Chicago, Illinois 60637, USA;
Committee on Immunology, Department of Pathology, The Howard Hughes Medical Institute, University of Chicago, Chicago, Illinois 60637, USA;
Committee on Immunology, Department of Pathology, The Howard Hughes Medical Institute, University of Chicago, Chicago, Illinois 60637, USA;
Committee on Immunology, Department of Pathology, The Howard Hughes Medical Institute, University of Chicago, Chicago, Illinois 60637, USA;
Committee on Immunology, Department of Pathology, The Howard Hughes Medical Institute, University of Chicago, Chicago, Illinois 60637, USA;
Committee on Immunology, Department of Pathology, The Howard Hughes Medical Institute, University of Chicago, Chicago, Illinois 60637, USA;
Department of Biology, Portland State University, Portland, Oregon 96207, USA;
Committee on Immunology, Department of Pathology, The Howard Hughes Medical Institute, University of Chicago, Chicago, Illinois 60637, USA;
机译:Cullin7 E3泛素连接酶的表征-cullin底物受体的异二聚化是调节cullin E3连接酶活性的新机制
机译:果蝇Cand1通过影响Cullin3的糖基化并控制Cullin3和衔接子蛋白的稳定性来调节Cullin3依赖的E3连接酶。
机译:果蝇Cand1通过影响Cullin3的糖基化并控制Cullin3和衔接子蛋白的稳定性来调节Cullin3依赖的E3连接酶
机译:鞘氨醇-1-磷酸是E3泛素连接酶TRAF2缺少的辅助因子
机译:Cullin 5 E3泛素连接酶:病毒和细胞功能
机译:BTB-ZF因素招募E3连接酶滞蛋白3以调节淋巴效应方案
机译:果蝇Cand1通过影响Cullin3的糖基化并控制Cullin3和衔接蛋白的稳定性来调节Cullin3依赖的E3连接酶