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首页> 外文期刊>Journal of Neurology >Apolipoprotein A5 gene C56G variant confers risk for the development of large-vessel associated ischemic stroke
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Apolipoprotein A5 gene C56G variant confers risk for the development of large-vessel associated ischemic stroke

机译:载脂蛋白A5基因C56G变异赋予大血管相关性缺血性卒中发展风险

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摘要

Ischemic stroke is a suddenly developing temporary or often permanent damage of the brain. Several candidate genes have been shown to have an impact in the pathogenesis of stroke. The aim of our study was to investigate the possible association between the C56G variant of the apolipoprotein A5 (APOA5) gene and ischemic stroke.
机译:缺血性中风是大脑突然发展的暂时性或经常性永久性损伤。已显示几种候选基因对中风的发病机制有影响。我们研究的目的是研究载脂蛋白A5(APOA5)基因的C56G变异与缺血性中风之间的可能联系。

著录项

  • 来源
    《Journal of Neurology》 |2008年第5期|649-654|共6页
  • 作者单位

    University of Pécs Dept. of Medical Genetics and Child Development Szigeti 12 7624 Pécs Hungary;

    University of Pécs Dept. of Medical Genetics and Child Development Szigeti 12 7624 Pécs Hungary;

    Dept. of Cerebrovascular Diseases Pándy Kálmán County Hospital Gyula Hungary;

    Central Laboratory Markusovszky County Hospital Szombathely Hungary;

    University of Pécs Dept. of Medical Genetics and Child Development Szigeti 12 7624 Pécs Hungary;

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  • 原文格式 PDF
  • 正文语种 eng
  • 中图分类
  • 关键词

    APOA5; C56G (S19W); ischemic stroke; susceptibility; IMT;

    机译:APOA5;C56G(S19W);缺血性中风;敏感性;IMT;

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