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机译:J774A.1巨噬细胞氧化铜纳米粒子诱导NLRP3炎性炎症的串联活化
Southwest Univ Coll Food Sci Minist Educ Key Lab Luminescence Anal & Mol Sensing Chongqing 400715 Peoples R China;
Southwest Univ Coll Food Sci Minist Educ Key Lab Luminescence Anal & Mol Sensing Chongqing 400715 Peoples R China;
Southwest Univ Coll Pharmaceut Sci Minist Educ Key Lab Luminescence Anal & Mol Sensing 2 Tiansheng Rd Chongqing 400715 Peoples R China;
Southwest Univ Coll Pharmaceut Sci Minist Educ Key Lab Luminescence Anal & Mol Sensing 2 Tiansheng Rd Chongqing 400715 Peoples R China;
Southwest Univ Coll Pharmaceut Sci Minist Educ Key Lab Luminescence Anal & Mol Sensing 2 Tiansheng Rd Chongqing 400715 Peoples R China|Chinese Acad Sci Res Ctr Ecoenvironm Sci State Key Lab Environm Chem & Ecotoxicol Beijing 100085 Peoples R China;
Copper oxide nanoparticles; Reactive oxygen species; Lysosomal damage; Cathepsin B; NLRP3 inflammasome;
机译:来自NLRP3和Caspase-1核小鼠的巨噬细胞表现出降低的炎症组活化,改变M1 / M2标记和氧化应激,ER应激和自噬基因的表达,响应于Iron:在铁介导的肝损伤中的NLRP3炎性作用。
机译:硫化氢通过在巨噬细胞Cys269的S-巯基-Mun衰减氧化应激诱导的NLRP3炎症组活化
机译:硫化氢通过在巨噬细胞Cys269的S-巯基C-Jun中衰减氧化应激诱导的NLRP3炎症体活化
机译:金属离子诱导的氧化应激会激活巨噬细胞中的炎性体吗?
机译:铜诱导的双氧和过氧化氢的活化,用于有机底物的脱氢和氧合
机译:氧化铜纳米粒子通过铜离子介导的血管内皮细胞中的P38 MAPK活化而诱导氧化性DNA损伤和细胞死亡。
机译:通过降低氧化应激,通过抑制巨噬细胞的Txnip依赖性NLRP3炎性激活来保护FlaVOnoid VI-16免受DSS诱导的结肠炎。