机译:脂多糖诱导的细菌易位是特定于肠道的,并与肠粘膜炎症相关。
Research Institute of General Surgery, Jinling Hospital, Medical School of Nanjing University, 305 East Zhongshan Road, Nanjing, 210002, Jiangsu Province, China;
Research Institute of General Surgery, Jinling Hospital, Medical School of Nanjing University, 305 East Zhongshan Road, Nanjing, 210002, Jiangsu Province, China;
Research Institute of General Surgery, Jinling Hospital, Medical School of Nanjing University, 305 East Zhongshan Road, Nanjing, 210002, Jiangsu Province, China;
Research Institute of General Surgery, Jinling Hospital, Medical School of Nanjing University, 305 East Zhongshan Road, Nanjing, 210002, Jiangsu Province, China;
Research Institute of General Surgery, Jinling Hospital, Medical School of Nanjing University, 305 East Zhongshan Road, Nanjing, 210002, Jiangsu Province, China;
lipopolysaccharide; bacterial translocation; tight junctions; intestinal permeability; endotoxemia;
机译:脂多糖诱导的细菌易位是肠道特异性的,并与肠粘膜炎症有关
机译:肠道菌群失调导致肠道粘膜屏障功能降低和细菌移位增加。
机译:肠缺血-再灌注损伤加剧了黄疸大鼠的肠粘膜损伤和细菌移位。
机译:粘膜免疫系统的反调节与粘膜炎症的机制
机译:前列腺素刺激缺血性猪肠道粘膜屏障功能恢复的机制:肠离子转运的作用
机译:特拉迪尔对大鼠肠缺血再灌注后肠黏膜屏障功能和细菌移位的影响
机译:肠道缺陷症有助于降低肠粘膜屏障功能和增加的细菌易位