Expression of 14-3-3 ζ and interaction with protein kinase C in the rat retina in early diabetes

Y. H. Kim; Y. S. Kim; S. S. Kang; H. S. Noh; H. J. Kim; G. J. Cho; W. S. Choi

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Expression of 14-3-3 ζ and interaction with protein kinase C in the rat retina in early diabetes

机译：糖尿病早期大鼠视网膜中14-3-3ζ的表达及其与蛋白激酶C的相互作用

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摘要

The present study aimed to investigate the expression levels of and the relationship between 14-3-3 ζ and protein kinase C (PKC) in the retina of early diabetes.

机译：本研究旨在探讨早期糖尿病视网膜中14-3-3ζ与蛋白激酶C（PKC）的表达水平及其之间的关系。

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来源
《Diabetologia》 |2005年第7期|1411-1415|共5页
作者
Y. H. Kim; Y. S. Kim; S. S. Kang; H. S. Noh; H. J. Kim; G. J. Cho; W. S. Choi;
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作者单位

Department of Anatomy and Neurobiology College of Medicine Institute of Health Science Gyeongsang National University;

Department of Anatomy and Neurobiology College of Medicine Institute of Health Science Gyeongsang National University;

Department of Anatomy and Neurobiology College of Medicine Institute of Health Science Gyeongsang National University;

Department of Anatomy and Neurobiology College of Medicine Institute of Health Science Gyeongsang National University;

Department of Anatomy and Neurobiology College of Medicine Institute of Health Science Gyeongsang National University;

Department of Anatomy and Neurobiology College of Medicine Institute of Health Science Gyeongsang National University;

Department of Anatomy and Neurobiology College of Medicine Institute of Health Science Gyeongsang National UniversityDepartment of Anatomy and Neurobiology College of Medicine Gyeongsang National University;

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正文语种 eng
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关键词
14-3-3 ζ; Diabetes; PKC activation; Retina; Streptozotocin;

机译：14-3-3ζ;糖尿病;PKC激活;视网膜;链脲佐菌素;

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1. Direct interaction between protein kinase C theta (PKC theta) and 14-3-3 tau in T cells: 14-3-3 overexpression results in inhibition of PKC theta translocation and function. [J] . N Meller, Y C Liu, T L Collins, Molecular and Cellular Biology . 1996,第10期

机译：T细胞中蛋白激酶C theta（PKC theta）和14-3-3 tau之间的直接相互作用：14-3-3过表达导致PKC theta易位和功能受到抑制。
2. Inactivation of 14-3-3 protein exacerbates cardiac hypertrophy and fibrosis through enhanced expression of protein kinase C beta2 in experimental diabetes. [J] . Gurusamy N, Watanabe K, Ma M, Biological & pharmaceutical bulletin . 2005,第6期

机译：14-3-3蛋白的失活通过增强蛋白激酶C beta2在实验性糖尿病中的表达而加剧心脏肥大和纤维化。
3. Anoxic preconditioning up-regulates 14-3-3 protein expression in neonatal rat cardiomyocytes through extracellular signal-regulated protein kinase 1/2 [J] . Chen HP, He M, Xu YL, Life sciences . 2007,第5期

机译：缺氧预处理通过细胞外信号调节蛋白激酶1/2上调新生大鼠心肌细胞中14-3-3蛋白的表达
4. Structural Protein Interactions Predict Kinase-Inhibitor Interactions in Upregulated Pancreas Tumour Genes Expression Data [C] . Gihan Dawelbait, Christian Pilarsky, Yanju Zhang, International Symposium on Computational Life Sciences(CompLife 2005); 20050925-27; Konstanz(DE) . 2005

机译：结构蛋白相互作用预测上调胰腺肿瘤基因表达数据中的激酶抑制剂相互作用。
5. Study of the interactions between mitogen-activated protein kinases and small molecule inhibitors of substrate proteins using fluorescence spectroscopy [D] . Nevels, Kerrick J. 2011

机译：利用荧光光谱研究促分裂原活化蛋白激酶与底物蛋白小分子抑制剂之间的相互作用
6. Direct interaction between protein kinase C theta (PKC theta) and 14-3-3 tau in T cells: 14-3-3 overexpression results in inhibition of PKC theta translocation and function. [O] . N Meller, Y C Liu, T L Collins, 1996

机译：T细胞中蛋白激酶C theta（PKC theta）和14-3-3 tau之间的直接相互作用：14-3-3过表达导致PKC theta易位和功能受到抑制。
7. Expression of 14-3-3 ζ and interaction with protein kinase C in the rat retina in early diabetes [O] . Y. H. Kim, Y. S. Kim, S. S. Kang, 2005

机译：14-3-3‰的表达与早期糖尿病大鼠视网膜蛋白激酶C的相互作用

Expression of 14-3-3 ζ and interaction with protein kinase C in the rat retina in early diabetes

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