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Nuclear Orphan Receptor TAK1/TR4-Deficient Mice Are Protected Against Obesity-Linked Inflammation, Hepatic Steatosis, and Insulin Resistance

机译:核孤儿受体TAK1 / TR4缺陷小鼠受到保护,防止肥胖相关的炎症,肝脂肪变性和胰岛素抵抗

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  • 来源
    《Diabetes》 |2011年第1期|p.177-188|共12页
  • 作者单位

    Hong Soon Kang,1 Kyoko Okamoto,1 Yong-Sik Kim,1 Yukimasa Takeda,1 Carl D. Bortner,2 Huaixin Dang,1 Taira Wada,3 Wen Xie,3 Xiao-Ping Yang,1 Grace Liao/ and Anton M. Jetten1From the 1 Cell Biology Section, Laboratory of Respiratory Biology, National Institute of Environmental Health Sciences, National Institutes of Health, Research Triangle Park, North Carolina, the 2 Laboratory of Signal Transduction, Division of Intramural Research, National Institute of Environmental Health Sciences, National Institutes of Health, Research Triangle Park, North Carolina, and the ''Center for Pharmacogenetics and Department of Pharmaceutical Sciences, University of Pittsburgh, Pittsburgh, Pennsylvania.Corresponding author: Anton M. Jetten, jetten@niehs.nih.gov.Received 4 May 2010 and accepted 14 September 2010. Published ahead of print at http,//diabetes.diabetesjournals.org on 23 September 2010. DOI: 10.2337/dbl0-0628.© 2011 by the American Diabetes Association. Readers may use this article as long as the work is properly cited, the use is educational and not for profit, and the work is not altered. See http://creativecommons.org/licenseS/by-nc-nd/3.0/ for details.The costs of publication of this article were defrayed in part, by ifie payment, of page chtuyes. This article must therefore be hereby marked "advertisement" in accordance with 18 U.S.C. Section 1734 solely to indicate this fact.,;

  • 收录信息 美国《科学引文索引》(SCI);美国《化学文摘》(CA);
  • 原文格式 PDF
  • 正文语种 eng
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  • 入库时间 2022-08-17 13:45:37

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