机译:核孤儿受体TAK1 / TR4缺陷小鼠受到保护,防止肥胖相关的炎症,肝脂肪变性和胰岛素抵抗
Cell Biology Section, Laboratory of Respiratory Biology, National Institute of Environmental Health Sciences, National Institutes of Health, Research Triangle Park, North Carolina;
Cell Biology Section, Laboratory of Respiratory Biology, National Institute of Environmental Health Sciences, National Institutes of Health, Research Triangle Park, North Carolina;
Cell Biology Section, Laboratory of Respiratory Biology, National Institute of Environmental Health Sciences, National Institutes of Health, Research Triangle Park, North Carolina;
Cell Biology Section, Laboratory of Respiratory Biology, National Institute of Environmental Health Sciences, National Institutes of Health, Research Triangle Park, North Carolina;
Laboratory of Signal Transduction, Division of Intramural Research, National Institute of Environmental Health Sciences, National Institutes of Health, Research Triangle Park, North Carolina;
Cell Biology Section, Laboratory of Respiratory Biology, National Institute of Environmental Health Sciences, National Institutes of Health, Research Triangle Park, North Carolina;
Center for Pharmacogenetics and Department of Pharmaceutical Sciences, University of Pittsburgh, Pittsburgh, Pennsylvania;
Center for Pharmacogenetics and Department of Pharmaceutical Sciences, University of Pittsburgh, Pittsburgh, Pennsylvania;
Cell Biology Section, Laboratory of Respiratory Biology, National Institute of Environmental Health Sciences, National Institutes of Health, Research Triangle Park, North Carolina;
Cell Biology Section, Laboratory of Respiratory Biology, National Institute of Environmental Health Sciences, National Institutes of Health, Research Triangle Park, North Carolina;
Cell Biology Section, Laboratory of Respiratory Biology, National Institute of Environmental Health Sciences, National Institutes of Health, Research Triangle Park, North Carolina;
机译:核孤儿受体TAK1 / TR4缺陷小鼠受到保护,防止肥胖相关的炎症,肝脂肪变性和胰岛素抵抗
机译:肝脏巨噬细胞的维生素D受体激活改善了小鼠肝脏炎症,脂肪变性和胰岛素抵抗力
机译:低密度脂蛋白受体相关蛋白1保护免受肝胰岛素抵抗和肝脂肪变性。
机译:孤儿核受体的激动剂:动物模型中培养细胞和HDL水平的诱导eAPO e合成之间的关系
机译:脂蛋白受体SR-B1缺乏增强脂肪组织炎症,并在小鼠饮食诱导的肥胖期间减少对肝脏脂肪变性的易感性
机译:核孤儿受体TAK1 / TR4缺陷小鼠受到保护防止肥胖相关的炎症肝脂肪变性和胰岛素抵抗
机译:核孤儿受体TAK1 / TR4缺陷小鼠受到保护,以防止肥胖相关的炎症,肝脂肪变性和胰岛素抵抗