机译:睫状神经营养因子通过PI3激酶依赖性途径刺激肌肉葡萄糖摄取,而该途径受肥胖影响
St. Vincent's Institute of Medical Research and Department of Medi-cine, University of Melbourne, Melbourne, Australia;
St. Vincent's Institute of Medical Research and Department of Medi-cine, University of Melbourne, Melbourne, Australia Department of Physiology, Monash University, Clayton, Victoria, Australia;
Ludwig Institute for Cancer Research, Parkville, Australia;
Institute of Diabe-tes, Obesity and Metabolism, University of Pennsylvania, Philadelphia, Pennsylvania;
St. Vincent's Institute of Medical Research and Department of Medi-cine, University of Melbourne, Melbourne, Australia CSIRO Molecular Health Technologies, Parkville, Australia;
St. Vincent's Institute of Medical Research and Department of Medi-cine, University of Melbourne, Melbourne, Australia Section of Human Physiology, Copenhagen Muscle Research Centre, Department of Exercise and Sport Sciences, University of Copen-hagen, Copenhagen, Denmark;
机译:睫状神经营养因子通过肥胖受损的PI3激酶依赖性途径刺激肌肉葡萄糖摄取。
机译:A-769662激活含AMPKβ1的配合物,但诱导葡萄糖摄取通过小鼠骨骼肌中的PI3-激酶依赖性途径。
机译:A-769662激活含AMPK beta1的复合物,但通过小鼠骨骼肌中的PI3激酶依赖性途径诱导葡萄糖摄取。
机译:绿茶的抗肥胖作用:可能参与调节葡萄糖摄取系统和抑制脂肪发生相关的转录因子
机译:瘦素对胰岛素刺激的骨骼肌葡萄糖摄取和心肌细胞增殖的影响。
机译:睫状神经营养因子通过肥胖受损的PI3激酶依赖性途径刺激肌肉葡萄糖摄取。
机译:睫状神经营养因子通过肥胖受损的PI3激酶依赖性途径刺激肌肉葡萄糖摄取。