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首页> 外文期刊>Journal of Rehabilitation Research and Development >A novel cadaveric model for anterior-inferior shoulder dislocation: Using forcible apprehension positioning
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A novel cadaveric model for anterior-inferior shoulder dislocation: Using forcible apprehension positioning

机译:前肩下脱位的新型尸体模型:使用强制性恐惧定位

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摘要

A novel cadaveric model for anteriorinferior shoulder dislocation using forcible apprehension positioning is presented. This model simulates an in vivo mechanism and yields capsulolabral lesions. The scapulae of 14 cadaveric entire upper limbs (82 ± 9 years, mean ± standard deviation) were each rigidly fixed to a custom shouldertesting device. A pneumatic system was used with pulleys and cables to simulate the rotator cuff and the deltoid muscles (anterior and middle portions). The glenohumeral joint was then positioned in the apprehension position of abduction, external rotation, and horizontal abduction. A 6degreeoffreedom load cell (Assurance Technologies, Garner, North Carolina) measured the joint reaction force that was then resolved into three orthogonal components of compression force, anteriorly directed force, and superiorly directed force. With the use of a thrust bearing, the humerus was moved along a rail with a servomotorcontrolled system at 50 mm/s that resulted in horizontal abduction. Force that developed passively in the pectoralis major muscle was recorded with an independent uniaxial load cell. Each of the glenohumeral joints dislocated anteriorinferior, six with avulsion of the capsulolabrum from the anteriorinferior glenoid bone and eight with capsulolabral stretching. Pectoralis major muscle force as well as the joint reaction force increased with horizontal abduction until dislocation. At dislocation, the magnitude of the pectoralis major muscle force, 609.6 N ± 65.2 N was similar to the compression force, 569.6 N ± 37.8 N. A cadaveric model yielded an anterior dislocation with a mechanism of forcible apprehension positioning when the appropriate shoulder muscles were simulated and a passive pectoralis major muscle was included. Capsulolabral lesions resulted, similar to those observed in vivo.
机译:提出了一种新的尸体模型,使用强制性的恐慌定位来治疗前下肩关节脱位。该模型模拟了体内机制,并产生了囊状纤维病变。将14个尸体整个上肢的肩cap骨(82±9年,平均±标准差)分别牢固地固定在定制的肩部测试设备上。气动系统与滑轮和电缆一起使用,以模拟肩袖和三角肌(前部和中部)。然后将盂肱关节置于外展,外旋和水平外展的拘束位置。一个6自由度的称重传感器(Assurance Technologies,加纳,北卡罗莱纳州)测量了关节反作用力,然后将其分解为压缩力,前向力和上方向力的三个正交分量。使用推力轴承,通过伺服电机控制系统以50 mm / s的速度使肱骨沿轨道移动,从而导致水平外展。用独立的单轴测力计记录了胸大肌被动发展的力。每个盂肱关节前下关节脱位,六个从肩盂前盂骨撕脱囊膜,八个在肩cap囊拉伸时脱臼。胸大肌和关节反作用力随着水平外展而增加直至脱位。脱位时,胸大肌力的大小为609.6 N±65.2 N,与压缩力为569.6 N±37.8 N相似。尸体模型产生前脱位,并且当适当的肩部肌肉处于适当位置时会产生强制性的positioning裂定位机制。模拟,包括被动胸大肌。发生了囊胚皮病变,与体内观察到的相似。

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