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机译:抗髓过氧化物酶抗体可增强多形核中性粒细胞的吞噬作用,IL-8产生和葡萄糖摄取,而不是抗蛋白酶3抗体,从而导致中性粒细胞活化诱导的细胞死亡
Department of Internal Medicine National Taiwan University Hospital National Taiwan University College of Medicine 7 Chung-Shan South Road Taipei Taiwan;
Department of Dermatology National Taiwan University Hospital National Taiwan University College of Medicine Taipei Taiwan;
Department of Laboratory Medicine Taipei Municipal Jen-Ai Hospital Taipei Taiwan;
Department of Internal Medicine Buddish Xindian-Tzu-Chi General Hospital Taipei Taiwan;
Department of Internal Medicine Buddish Xindian-Tzu-Chi General Hospital Taipei Taiwan;
Department of Internal Medicine National Taiwan University Hospital National Taiwan University College of Medicine 7 Chung-Shan South Road Taipei Taiwan;
Section of Allergy Immunology and Rheumatology Veterans General Hospital Taipei Taiwan;
Department of Internal Medicine National Taiwan University Hospital National Taiwan University College of Medicine 7 Chung-Shan South Road Taipei Taiwan;
Activation-induced cell death; Anti-neutrophil cytoplasmic autoantibody; Myeloperoxidase; Polymorphonuclear neutrophil; Proteinase-3;
机译:抗髓过氧化物酶抗体可增强多形核中性粒细胞的吞噬作用,IL-8产生和葡萄糖摄取,而不是抗蛋白酶3抗体,从而导致活化诱导的中性粒细胞死亡。
机译:抗髓过氧化物酶抗体可增强多形核中性粒细胞的吞噬作用,IL-8产生和葡萄糖摄取,而不是抗蛋白酶3抗体,从而导致活化诱导的中性粒细胞死亡。
机译:抗中性粒细胞胞浆抗体和效应器CD4 +细胞在抗髓过氧化物酶新月型肾小球肾炎中起非冗余作用。
机译:在多核核心子粒细胞上表达的触发受体(TREM)-1表达增强了与TLR4的协同作用中鼻子的细菌间隙
机译:IgM抗体可增强未成熟树突状细胞对凋亡细胞的吞噬作用。
机译:抗CD45亚型抗体通过蛋白酪氨酸磷酸化和p56lck酪氨酸激酶的差异抑制来增强中性粒细胞的吞噬作用和IL-8和TNF-α的基因表达
机译:抗CD45亚型抗体通过蛋白酪氨酸磷酸化和p56lck酪氨酸激酶的差异抑制来增强中性粒细胞的吞噬作用和IL-8和TNF-α的基因表达