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机译:ECT2基因的功能异常可能导致肾小管间质损伤,导致局灶节段性肾小球硬化
Department of Pediatrics, Kinki University School of Medicine, 377-2 Ohno-higashi, Osaka-Sayama, 589-8511, Japan;
Department of Pediatrics, Kinki University School of Medicine, 377-2 Ohno-higashi, Osaka-Sayama, 589-8511, Japan;
Department of Pediatrics, Kinki University School of Medicine, 377-2 Ohno-higashi, Osaka-Sayama, 589-8511, Japan;
Department of Pediatrics, Kinki University School of Medicine, 377-2 Ohno-higashi, Osaka-Sayama, 589-8511, Japan;
Department of Pediatrics, Kinki University School of Medicine, 377-2 Ohno-higashi, Osaka-Sayama, 589-8511, Japan;
Department of Pediatrics, Kinki University School of Medicine, 377-2 Ohno-higashi, Osaka-Sayama, 589-8511, Japan;
Department of Pediatrics, Kinki University School of Medicine, 377-2 Ohno-higashi, Osaka-Sayama, 589-8511, Japan;
Hyperfiltration; Tight junction; Tubular epithelial cell; Tubulointerstitial injury;
机译:ECT2基因的功能异常可能导致肾小管间质损伤,导致局灶节段性肾小球硬化。
机译:ECT2基因的功能异常可能导致肾小管间质损伤,导致局灶节段性肾小球硬化。
机译:早期局灶节段性肾小球硬化是肾移植原发性无功能的原因
机译:局灶性节段性肾小球粥样硬化的发病机制与临床意义
机译:局灶节段性肾小球硬化中足细胞的遗传和细胞研究
机译:ECT2基因的功能异常可能导致肾小管间质损伤导致局灶节段性肾小球硬化
机译:ECT2基因的功能异常可能导致肾小管间质损伤,导致局灶节段性肾小球硬化