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机译:Wnt和前列腺素E 2 sub>在胃癌发生中的协同作用触发CD44 + sup>慢循环肿瘤细胞扩增
Division of Gene Regulation, Institute for Advanced Medical Research, School of Medicine, Keio University, Shinjuku-ku, Tokyo, and Core Research for Evolutional Science and Technology (CREST), Japan Science and Technology Agency, Tokyo|Department of Gastroenterological Surgery, Graduate School of Medical Science, Kumamoto University, Kumamoto;
Division of Genetics, Cancer Research Institute, Kanazawa University, Kanazawa;
Division of Genetics, Cancer Research Institute, Kanazawa University, Kanazawa;
Division of Gene Regulation, Institute for Advanced Medical Research, School of Medicine, Keio University, Shinjuku-ku, Tokyo, and Core Research for Evolutional Science and Technology (CREST), Japan Science and Technology Agency, Tokyo;
Cell Biology Laboratory, Department of Pharmaceutical Sciences, School of Pharmacy, Kinki University, Higashiosaka-shi, Osaka, Japan;
Cell Biology Laboratory,;
机译:Wnt和前列腺素E2在胃癌发生中的协同作用触发了CD44 +慢循环肿瘤细胞的扩增。
机译:Wnt和前列腺素E 2 SUB>在胃癌发生中的协同作用触发CD44 + SUP>慢循环肿瘤细胞的扩增
机译:CD44 + / CD24-肿瘤干细胞的增多以及COX-2和HDAC6的上调是HER2在乳腺癌发生中的主要功能
机译:COX-2 / PGE_2在小鼠胃肿瘤中的信号和传染性刺激
机译:刺猬-Wnt病理上皮芽发育和皮肤肿瘤发生过程中的相互作用。
机译:Notch和Wnt信号共同控制肠道中的细胞增殖和肿瘤发生
机译:CD44 +缓慢循环肿瘤细胞膨胀是通过WNT和前列腺素E2胃肿瘤的合作作用引发的
机译:微小RNa调节CD44 +前列腺肿瘤干/祖细胞和前列腺癌发展/转移。