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机译:NLRP3炎性体激活的机制
Inflammation Program, University of Iowa, Iowa City, Iowa,Department of Internal Medicine, University of Iowa, Iowa City, Iowa,Veterans Affairs Medical Center, Iowa City, Iowa,University of Iowa, 2501 Crosspark Road, D156 MTF, Coralville, IA 52241;
Inflammation Program, University of Iowa, Iowa City, Iowa;
Inflammation Program, University of Iowa, Iowa City, Iowa,Department of Internal Medicine, University of Iowa, Iowa City, Iowa;
NLRP3; inflammasome; caspase-1; mitochondria; calcium;
机译:NLRP3炎症组合的机制:NLRP3吡林结构域的三聚化但未分化诱导IL-1β的鲁棒激活
机译:关于NLRP3炎症体激活的调控机制的最新方法
机译:NLRP3炎症组活化剂和灭活剂的分子机制进展
机译:使用同位素编码的ATP - 亲和探针和靶向质谱法在NLRP3炎症组活化期间洞察Kinome扰动
机译:金黄色葡萄球菌白毛虫的机制和后果AB介导的宿主NLRP3炎症的活化
机译:血红素和钴原卟啉通过增强自噬抑制NLRP3炎性体的激活:炎性体调控的新机制。
机译:NLRP3炎症:激活和调节机制概述