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机译:通过在蛋白质的N末端添加糖基化位点来修饰凝血因子VIII的表达
Institute for Experimental Hematology and Transfusion Medicine University Clinics Bonn University of Bonn Sigmund-Freud-Str. 25 53105 Bonn Germany;
Institute for Experimental Hematology and Transfusion Medicine University Clinics Bonn University of Bonn Sigmund-Freud-Str. 25 53105 Bonn Germany;
Institute for Experimental Hematology and Transfusion Medicine University Clinics Bonn University of Bonn Sigmund-Freud-Str. 25 53105 Bonn Germany;
Institute for Experimental Hematology and Transfusion Medicine University Clinics Bonn University of Bonn Sigmund-Freud-Str. 25 53105 Bonn Germany;
Institute for Experimental Hematology and Transfusion Medicine University Clinics Bonn University of Bonn Sigmund-Freud-Str. 25 53105 Bonn Germany;
Institute for Experimental Hematology and Transfusion Medicine University Clinics Bonn University of Bonn Sigmund-Freud-Str. 25 53105 Bonn Germany;
Institute for Experimental Hematology and Transfusion Medicine University Clinics Bonn University of Bonn Sigmund-Freud-Str. 25 53105 Bonn Germany;
Glycosylation; Factor VIII; Protein secretion; BiP; Calnexin; Calreticulin;
机译:在凝血因子VIII中产生异位N-糖基化位向的畸形突变的分子机制
机译:A2结构域的N-糖基化位点附近的畸形突变导致凝血因子VIII中的各种细胞内运输缺陷
机译:凝血因子VIII中共享非常低密度脂蛋白受体和低密度脂蛋白受体相关蛋白的结合位点。
机译:赖氨酸残基在血液凝固因子VIII与其间隙受体低密度脂蛋白受体相关蛋白的作用
机译:组蛋白修饰蛋白和染色质重塑因子在猪卵母细胞和发育中的胚胎中的表达。
机译:A2结构域N-糖基化位点附近的错义突变导致凝血因子VIII中的各种细胞内运输缺陷
机译:A2结构域N-糖基化位点附近的错义突变导致凝血因子VIII中的各种细胞内运输缺陷
机译:因子VIII / von Willebrand因子蛋白(F VIII / vWF)和F VIII / vWF-差冷沉淀物对中枢神经系统缺血后微血管再灌注的影响