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机译:STAT3,p38 MAPK和NF-kB在结核病中驱动非对抗性单核细胞依赖性成纤维细胞MMP-1分泌
Department of Infectious Diseases and Immunity, Hammersmith Campus, Imperial College, London,United Kingdom;
Department of Infectious Diseases and Immunity, Hammersmith Campus, Imperial College, London,United Kingdom;
Department of Infectious Diseases and Immunity, Hammersmith Campus, Imperial College, London,United Kingdom;
Department of Microbiology, Universidad Peruana Cayetano Heredia, Lima, Peru;
Department of Microbiology, Universidad Peruana Cayetano Heredia, Lima, Peru;
Department of Microbiology, Universidad Peruana Cayetano Heredia, Lima, Peru ,Department of International Health, Johns Hopkins University, School of Hygiene and Public Health, Baltimore, Maryland;
Department of Histopathology, Hammersmith Campus, Imperial College, London, United Kingdom;
Department of Infectious Diseases and Immunity, Hammersmith Campus, Imperial College, London,United Kingdom;
stromal cells; monocytes/macrophages; human; signal transduction; inflammation;
机译:多吡吡吡吡吡吡啶鎓酰基酸促进白细胞介素-6,并通过JAK / STAT3和P38 MAPK信号转导途径促进白细胞介素-8在人皮肤成纤维细胞中分泌
机译:单核细胞依赖性成纤维细胞CXCL8分泌发生在结核病中,并限制了分枝杆菌在巨噬细胞内的存活。
机译:美伐他汀通过p38MAPK和NF-kB分泌骨保护素
机译:6 -Igngingerol对小鼠皮肤中PMA诱导的COX-2表达和NF-KB和P38 MAPK的抑制作用
机译:在人肺和皮肤成纤维细胞中研究了炎症和氧化应激信号转导中的MAPK ERK,JNK和P38。
机译:STAT3p38 MAPK和NF-κB促进结核病中对立的单核细胞依赖性成纤维细胞MMP-1分泌
机译:STAT3,p38 MAPK和NF-κB促进结核病中对立的单核细胞依赖性成纤维细胞MMP-1分泌