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机译:通过消融前列腺素E2受体亚型EP1或EP2来防止Toll样受体4依赖性先天性免疫激活对海马神经发生的保护。
From the Departments of Pathology,University of Washington, Seattle, Washington;
From the Departments of Pathology,University of Washington, Seattle, Washington;
From the Departments of Pathology,University of Washington, Seattle, Washington;
From the Departments of Pathology,University of Washington, Seattle, Washington;
and Neurological Surgery,University of Washington, Seattle, Washington;
From the Departments of Pathology,University of Washington, Seattle, Washington;
and the Department of Medicine,Vanderbilt University, Nashville, Tennessee;
and Neurological Surgery,University of Washington, Seattle, Washington;
and Neurological Surgery,University of Washington, Seattle, Washington;
From the Departments of Pathology,University of Washington, Seattle, Washington;
机译:通过消融前列腺素E2受体亚型EP1或EP2,保护海马神经元免受toll样受体4依赖性先天性免疫激活的影响。
机译:锰曝光引起了前列腺素E2受体EP1和EP2中下丘脑中丘脑中患者激活GnRH分泌的雄性小鼠的生殖毒性
机译:NSAIDs和前列腺素E2受体EP2的消除可抑制CD14依赖性先天免疫介导的体内神经元损伤。
机译:EP受体亚型 - 前列腺素e2〜a调节免疫细胞功能的依赖性
机译:人类EP1前列腺素受体激活新的信号转导途径:前列腺素E2的G蛋白偶联受体。
机译:消融前列腺素E2受体亚型EP1或EP2防止Toll样受体4依赖性先天免疫激活海马神经发生。
机译:消融前列腺素E2受体亚型EP1或EP2,防止Toll样受体4依赖性先天免疫激活海马神经发生。
机译:对于肺炎链球菌的先天免疫和1型体液免疫均由myD88介导,但它们对Toll样受体2的相对依赖性水平不同