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Pharyngeal Microflora Disruption by Antibiotics Promotes Airway Hyperresponsiveness after Respiratory Syncytial Virus Infection

机译：呼吸道合胞病毒感染后，抗生素破坏咽部菌群可促进气道高反应性。

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BackgroundRegulatory T cells (Treg cells), which are essential for regulation of immune response to respiratory syncytial virus (RSV) infection, are promoted by pharyngeal commensal pneumococcus. The effects of pharyngeal microflora disruption by antibiotics on airway responsiveness and relative immune responses after RSV infection have not been clarified.

机译：背景技术咽部共生肺炎球菌可促进调节性T细胞（Treg细胞）对调节呼吸道合胞病毒（RSV）感染的免疫反应至关重要。尚不清楚抗生素对咽部菌群的破坏对RSV感染后气道反应性和相对免疫反应的影响。

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期刊名称 PLoS Clinical Trials
作者
Ke Ni; Simin Li; Qiuling Xia; Na Zang; Yu Deng; Xiaohong Xie; Zhengxiu Luo; Yan Luo; Lijia Wang; Zhou Fu; Enmei Liu;
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年(卷),期 2009(7),7
年度 2009
页码 e41104
总页数 9
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专利

1. Alteration of airway neuropeptide expression and development of airway hyperresponsiveness following respiratory syncytial virus infection. [J] . Dakhama A, Park JW, Taube C, American Journal of Physiology . 2005,第4期

机译：呼吸道合胞病毒感染后气道神经肽表达的改变和气道高反应性的发展。
2. Alteration of airway neuropeptide expression and development of airway hyperresponsiveness following respiratory syncytial virus infection. [J] . Dakhama A, Park JW, Taube C, American Journal of Physiology . 2005,第4期

机译：呼吸道合胞病毒感染后气道神经肽表达的改变和气道高反应性的发展。
3. Critical Roles for Interleukin - 4 and Interleukin -5 during Respiratory Syncytial Virus Infection in the Development of Airway Hyperresponsiveness after Airway Sensitization [J] . Jurgen Schwarze, Grzegorz Cieslewicz, Anthony Joetham American journal of respiratory and critical care medicine . 2000,第2期

机译：呼吸道合胞病毒感染期间白细胞介素-4和白细胞介素-5在气道敏化后气道高反应性发展中的关键作用
4. PPARgamma- and Toll-Like Receptor-2 Agonists Might Have Therapeutical Value in Respiratory Syncytial Virus (RSV)-Induced Airway Diseases PPARgamma Agonists May Act in a PPARgamma-Independent Manner [C] . R. Arnold, W. Konig Collegium Internationale Allergologicum . 2007

机译：pParγ-和Toll样受体-2激动剂可能对呼吸道合胞病毒（RSV）的治疗值（RSV）诱导的气道疾病Pparγ激动剂可以以ppargamma独立的方式行事
5. Immunopathogenesis of bovine respiratory syncytial virus and acute bovine viral diarrhea virus infections in calves [D] . Brodersen, Bruce Wesley. 1996

机译：牛犊牛呼吸道合胞病毒和急性牛病毒性腹泻病毒感染的免疫发病机制
6. Pulmonary C Fibers Modulate MMP-12 Production via PAR2 and Are Involved in the Long-Term Airway Inflammation and Airway Hyperresponsiveness Induced by Respiratory Syncytial Virus Infection [O] . Na Zang, Jianguo Zhuang, Yu Deng, 2016

机译：肺C纤维通过PAR2调节MMP-12的产生并参与呼吸道合胞病毒感染引起的长期气道炎症和气道高反应性。
7. Pharyngeal microflora disruption by antibiotics promotes airway hyperresponsiveness after respiratory syncytial virus infection. [O] . Ke Ni, Simin Li, Qiuling Xia, 2012

机译：在呼吸道合胞病毒感染后，抗生素导致的咽部微生物群落破坏促进气道高反应性。

Pharyngeal Microflora Disruption by Antibiotics Promotes Airway Hyperresponsiveness after Respiratory Syncytial Virus Infection

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