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Nitric oxide protects the heart from ischemia-induced apoptosis and mitochondrial damage via protein kinase G mediated blockage of permeability transition and cytochrome c release

机译：一氧化氮通过蛋白激酶G介导的通透性转变和细胞色素c释放的阻滞保护心脏免受缺血诱导的细胞凋亡和线粒体损害

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摘要

BackgroundHeart ischemia can rapidly induce apoptosis and mitochondrial dysfunction via mitochondrial permeability transition-induced cytochrome c release. We tested whether nitric oxide (NO) can block this damage in isolated rat heart, and, if so, by what mechanisms.

机译：背景心脏缺血可通过线粒体通透性转变诱导的细胞色素C释放迅速诱导细胞凋亡和线粒体功能障碍。我们测试了一氧化氮（NO）是否可以阻止离体大鼠心脏的这种损害，如果可以，则通过什么机制来阻断。

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期刊名称 Journal of Biomedical Science
作者
Vilmante Borutaite; Ramune Morkuniene; Odeta Arandarcikaite; Aiste Jekabsone; Jurgita Barauskaite; Guy C Brown;
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作者单位

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年(卷),期 2009(16),1
年度 2009
页码 70
总页数 12
原文格式 PDF
正文语种
中图分类医学史;
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1. Nitric oxide protects the heart from ischemia-induced apoptosis and mitochondrial damage via protein kinase G mediated blockage of permeability transition and cytochrome c release [J] . Vilmante Borutaite, Ramune Morkuniene, Odeta Arandarcikaite, Journal of biomedical science. . 2009,第1期

机译：一氧化氮通过蛋白激酶G介导的通透性转变和细胞色素C释放的阻断作用，保护心脏免受缺血诱导的细胞凋亡和线粒体损害。
2. Nitric oxide protects the heart from ischemia-induced apoptosis and mitochondrial damage via protein kinase G mediated blockage of permeability transition and cytochrome c release [J] . Vilmante Borutaite, Ramune Morkuniene, Odeta Ararcikaite, Journal of biomedical science. . 2009,第1期

机译：通过蛋白激酶G介导的渗透性转变和细胞色素C释放，一氧化氮保护心脏免受缺血诱导的细胞凋亡和线粒体损伤的影响
3. Nitric oxide protects the heart from ischemia-induced apoptosisand mitochondrial damage via protein kinase G mediated blockageof permeability transition and cytochrome c release [J] . Vilmante Borutaite, Ramune Morkuniene, Odeta Arandarcikaite, Journal of biomedical science. . 2009,第8期

机译：一氧化氮通过蛋白激酶G介导的通透性转变和细胞色素C释放的阻滞保护心脏免受缺血诱导的细胞凋亡和线粒体损害
4. Effects of Protein Kinase C Selective Beta II Peptide Inhibitor on Real-Time Blood Nitric Oxide and Hydrogen Peroxide Release Under Acute Hyperglycemia [C] . Michael Minni, Qian Chen, Kyle Bartol American Peptide Symposium . 2011

机译：蛋白激酶C选择性βII肽抑制剂对急性高血糖血症实时血液一氧化氮和过氧化氢释放的影响
5. Hindbrain 5'-Monophosphate Activated Protein Kinase Mediates Inhibitory Effects of Short Term Food Deprivation in the Gonadotropin Releasing Hormone Luteinizing Hormone Axis: Role of Noradrenergic, Nitric Oxide and Opioid Neurons [D] . Shakya, Manita. 2018

机译：Hindbrain 5'-单磷酸盐激活的蛋白激酶介导促性腺激素释放激素促黄体生成激素轴的短期食物剥夺的抑制作用：去甲肾上腺素能，一氧化氮和阿片类神经元的作用
6. Nitric oxide-induced eosinophil apoptosis is dependent on mitochondrial permeability transition (mPT) JNK and oxidative stress: apoptosis is preceded but not mediated by early mPT-dependent JNK activation [O] . Pinja Ilmarinen-Salo, Eeva Moilanen, Vuokko L Kinnula, 2012

机译：一氧化氮诱导的嗜酸性粒细胞凋亡取决于线粒体通透性转变（mPT）JNK和氧化应激：细胞凋亡先于但不由早期mPT依赖性JNK激活介导
7. Nitric oxide protects the heart from ischemia-induced apoptosis and mitochondrial damage via protein kinase G mediated blockage of permeability transition and cytochrome c release [O] . Borutaite Vilmante, Morkuniene Ramune, Arandarcikaite Odeta, 2011

机译：一氧化氮通过蛋白激酶G介导的通透性转变和细胞色素c释放的阻滞保护心脏免受缺血诱导的细胞凋亡和线粒体损害

Nitric oxide protects the heart from ischemia-induced apoptosis and mitochondrial damage via protein kinase G mediated blockage of permeability transition and cytochrome c release

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