首页> 美国卫生研究院文献>The Journal of Biological Chemistry >Withdrawal: A novel human phosphatidylethanolamine-binding protein resists tumor necrosis factor α-induced apoptosis by inhibiting mitogen-activated protein kinase pathway activation and phosphatidylethanolamine externalization.

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Withdrawal: A novel human phosphatidylethanolamine-binding protein resists tumor necrosis factor α-induced apoptosis by inhibiting mitogen-activated protein kinase pathway activation and phosphatidylethanolamine externalization.

机译：提取：一种新型人磷脂酰乙醇胺结合蛋白通过抑制丝分裂活性蛋白激酶途径和磷脂酰乙醇胺外化来抵抗肿瘤坏死因子α-诱导的细胞凋亡。

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VOLUME 279 (2004) PAGES 45855-45864

机译：第279卷（2004）第45855-45864页

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期刊名称 The Journal of Biological Chemistry
作者
Xiaojian Wang; Nan Li; Bin Liu; Hongying Sun; Taoyong Chen; Hongzhe Li; Jianming Qiu; Lihuang Zhang; Tao Wan; Xuetao Cao;
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作者单位

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年(卷),期 2020(295),39
年度 2020
页码 13692
总页数 1
原文格式 PDF
正文语种
中图分类分子生物学;
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专利

1. Flavopiridol suppresses tumor necrosis factor-induced activation of activator protein-1, c-Jun N-terminal kinase, p38 mitogen-activated protein kinase (MAPK), p44/p42 MAPK, and Akt, inhibits expression of antiapoptotic gene products, and enhances apoptosis through cytochrome c release and caspase activation in human myeloid cells (Retraction of vol 73, pg 1549, 2008) [J] . Takada Y., Sethi G., Sung B., Molecular pharmacology. . 2016,第1期

机译：黄酮哌啶醇抑制肿瘤坏死因子诱导的活化蛋白1，c-Jun N端激酶，p38丝裂原活化蛋白激酶（MAPK），p44 / p42 MAPK和Akt的激活，抑制抗凋亡基因产物的表达并增强凋亡通过人类髓样细胞中细胞色素c的释放和胱天蛋白酶的活化（Retract of vol 73，pg 1549，2008）
2. SODIUM SALICYLATE INDUCES APOPTOSIS VIA P38 MITOGEN-ACTIVATED PROTEIN KINASE BUT INHIBITS TUMOR NECROSIS FACTOR-INDUCED C-JUN N-TERMINAL KINASE STRESS-ACTIVATED PROTEIN KINASE ACTIVATION [J] . Schwenger P., Vietor I., Basilico C., Proceedings of the National Academy of Sciences of the United States of America . 1997,第7期

机译：水杨酸钠通过P38丝裂原活化的蛋白激酶诱导细胞凋亡，但抑制肿瘤坏死因子诱导的C-Jun N末端激酶应力激活的蛋白激酶活化。
3. Sodium salicylate induces apoptosis via p38 mitogen-activated protein kinase but inhibits tumor necrosis factor-induced c-Jun N-terminal kinase/stress-activated protein kinase activation [J] . Paul Schwenger, Paola Bellosta, Ilja Vietor Proceedings of the National Academy of Sciences of the United States of America . 1997,第7期

机译：水杨酸钠通过p38丝裂原激活的蛋白激酶诱导细胞凋亡，但抑制肿瘤坏死因子诱导的c-Jun N端激酶/应激激活的蛋白激酶激活
4. Activation of Mitogen-activated Protein Kinases and Protein Kinase B/akt Signaling By Oxidative Stress in Vascular Smooth Muscle Cells: involvement in Vascular Pathophysiology [C] . Ashok K. Srivastava, Nihar R. Pandey, Antoine Blanc International Society for Heart Research.Congress . 2004

机译：血管平滑肌细胞氧化应激激活丝裂剂活化蛋白激酶和蛋白激酶B / Akt信号传导：血管病理生理学中的参与
5. Impact of Hepatitis C Virus Core Protein on the Tumor Necrosis Factor Apoptosis-inducing Ligand Pathway in Peripheral Blood Mononuclear Cells [D] . Guy, Janelle. 2018

机译：丙型肝炎病毒核心蛋白对外周血单个核细胞中肿瘤坏死因子凋亡诱导配体途径的影响
6. Re: Takada Y Sethi G Sung B and Aggarwal BB (2008) Flavopiridol suppresses tumor necrosis factor-induced activation of activator protein-1 c-Jun N-terminal kinase p38 mitogen-activated protein kinase (MAPK) p44/p42 MAPK and Akt inhibits expression of antiapoptotic gene products and enhances apoptosis through cytochrome c release and caspase activation in human myeloid cells Mol Pharmacol May 2008 73:1549–1557; doi:10.1124/mol.107.041350 [O] . -1

机译：回复：Takada YSethi GSung B和Aggarwal BB（2008）黄酮哌啶醇可抑制肿瘤坏死因子诱导的活化蛋白1c-Jun N端激酶p38丝裂原活化蛋白激酶（MAPK）p44活化/ p42 MAPK和Akt可以抑制抗凋亡基因产物的表达并通过细胞色素c释放和胱天蛋白酶在人骨髓细胞中的活化来增强细胞凋亡Mol Pharmacol2008年5月73：1549-1557； doi：10.1124 / mol.107.041350
7. A Novel Human Phosphatidylethanolamine-binding Protein Resists Tumor Necrosis Factor α-induced Apoptosis by Inhibiting Mitogen-activated Protein Kinase Pathway Activation and Phosphatidylethanolamine Externalization [O] . Xiaojian Wang, Nan Li, Bin Liu, 2004

机译：一种新型人磷脂酰乙醇胺结合蛋白通过抑制丝裂原激活的蛋白激酶途径和磷脂酰乙醇胺外化来抵抗肿瘤坏死因子α-诱导的细胞凋亡
8. Hantaan Virus Nucleocapsid Protein Binds to Importin alpha Proteins and Inhibits Tumor Necrosis Factor Alpha-Induced Activation of Nuclear Factor Kappa B [R] . Taylor, S. L., Frias-Staheli, N., Garcia-Sastre, A., 2008

机译：汉坦病毒核衣壳蛋白与进口蛋白α蛋白结合并抑制肿瘤坏死因子α诱导的核因子κB活化

Withdrawal: A novel human phosphatidylethanolamine-binding protein resists tumor necrosis factor α-induced apoptosis by inhibiting mitogen-activated protein kinase pathway activation and phosphatidylethanolamine externalization.

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