首页> 美国卫生研究院文献>Journal of Experimental Clinical Cancer Research : CR >Calycosin suppresses TGF-β-induced epithelial-to-mesenchymal transition and migration by upregulating BATF2 to target PAI-1 via the Wnt and PI3K/Akt signaling pathways in colorectal cancer cells

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Calycosin suppresses TGF-β-induced epithelial-to-mesenchymal transition and migration by upregulating BATF2 to target PAI-1 via the Wnt and PI3K/Akt signaling pathways in colorectal cancer cells

机译：Calycosin通过经由Wnt和PI3K / Akt信号通路上调BATF2到靶PAI-1上调大肠癌细胞中的TGF-β诱导的上皮向间充质转化和迁移。

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摘要

ObjectivesTo determine whether the upregulation of basic leucine zipper ATF-like transcription factor 2 (BATF2) by calycosin suppresses the growth and epithelial-to-mesenchymal transition (EMT) in human colorectal cancer (CRC) cells.

机译：目的确定花胶苷是否上调碱性亮氨酸拉链ATF样转录因子2（BATF2）抑制人类结直肠癌（CRC）细胞的生长和上皮间质转化（EMT）。

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期刊名称 Journal of Experimental Clinical Cancer Research : CR
作者
Qun Wang; Weijun Lu; Tao Yin; Li Lu;
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作者单位

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年(卷),期 2019(38),-1
年度 2019
页码 240
总页数 12
原文格式 PDF
正文语种
中图分类肿瘤学;
关键词
BATF2 Calycosin Cell migration Colorectal cancer PAI-1;

机译：BATF2;Calycosin;细胞迁移;结肠直肠癌;PAI-1;

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1. Alisertib induces cell cycle arrest and autophagy and suppresses epithelial-to-mesenchymal transition involving PI3K/Akt/mTOR and sirtuin 1-mediated signaling pathways in human pancreatic cancer cells [J] . Feng Wang, Hai Li, Xiao-Gang Yan, Drug Design, Development and Therapy . 2015,第5期

机译：Alisertib诱导人胰腺癌细胞中涉及PI3K / Akt / mTOR和sirtuin 1介导的信号通路的细胞周期停滞和自噬并抑制上皮向间质转化
2. Manganese‐12 acetate suppresses the migration, invasion, and epithelial–mesenchymal transition by inhibiting Wnt/β‐catenin and PI3K/AKT signaling pathways in breast cancer cells [J] . Hongping Ju, Yongxia Li, Xiqian Xing, Thoracic cancer. . 2018,第3期

机译：乙酸锰12通过抑制乳腺癌细胞中的Wnt /β-catenin和PI3K / AKT信号通路来抑制迁移，侵袭和上皮间质转化
3. Calycosin induces apoptosis in colorectal cancer cells, through modulating the ER beta/MiR-95 and IGF-1R, PI3K/Akt signaling pathways [J] . Zhao Xinge, Li Xin, Ren Qianyao, Gene: An International Journal Focusing on Gene Cloning and Gene Structure and Function . 2016,第1期

机译：Calycosin通过调节ER beta / MiR-95和IGF-1R，PI3K / Akt信号通路诱导大肠癌细胞凋亡
4. Inspecting the Role of PI3K/AKT Signaling Pathway in Cancer Development Using an In Silico Modeling and Simulation Approach [C] . Pedro Pablo Gonzalez-Perez, Maura Cardenas-Garcia International work-conference on bioinformatics and biomedical engineering . 2018

机译：使用计算机模拟和仿真方法检查PI3K / AKT信号通路在癌症发展中的作用
5. The multi-targeted receptor tyrosine kinase inhibitor, linifanib (ABT-869), induces apoptosis and inhibits proliferation of leukemia cells through phosphoinositide-3 kinase (PI3K)/AKT-dependent signaling pathways. [D] . Hernandez, Jenny Elizabeth. 2010

机译：多靶点受体酪氨酸激酶抑制剂利尼法尼（ABT-869）通过磷酸肌醇3激酶（PI3K）/ AKT依赖性信号传导途径诱导凋亡并抑制白血病细胞的增殖。
6. Correction to: Calycosin suppresses TGF-β-induced epithelial-to-mesenchymal transition and migration by upregulating BATF2 to target PAI-1 via the Wnt and PI3K/Akt signaling pathways in colorectal cancer cells [O] . Qun Wang, Weijun Lu, Tao Yin, 2019

机译：校正至：Calycosin通过经由Wnt和PI3K / Akt信号传导途径上调大肠癌细胞中的BATF2使其靶向PAI-1从而抑制TGF-β诱导的上皮向间充质转化和迁移。
7. Calycosin suppresses TGF-β-induced epithelial-to-mesenchymal transition and migration by upregulating BATF2 to target PAI-1 via the Wnt and PI3K/Akt signaling pathways in colorectal cancer cells [O] . Qun Wang, Weijun Lu, Tao Yin, 2019

机译：钙霉素抑制TGF-β-诱导的上皮 - 间充质转变，并通过在结肠直肠癌细胞中通过WNT和PI3K / AKT信号传导途径将BAIF2缩减至靶PAI-1来迁移

Calycosin suppresses TGF-β-induced epithelial-to-mesenchymal transition and migration by upregulating BATF2 to target PAI-1 via the Wnt and PI3K/Akt signaling pathways in colorectal cancer cells

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