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Effects of Botulinum Toxin on Reducing the Co-contraction of Antagonists in Birth Brachial Plexus Palsy

机译:肉毒杆菌毒素对减少臂丛神经麻痹拮抗剂拮抗作用的影响

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摘要

Birth brachial plexus palsy (BBPP) is usually caused by plexus traction during difficult delivery. Although the possibility of complete recovery is relatively high, 5% to 25% of BBPP cases result in prolonged and persistent disability. In particular, muscle imbalance and co-contraction around the shoulder and elbow cause abnormal motor performance, osseous deformities, and joint contracture. Physical and occupational therapies have most commonly been used, but these conventional therapeutic strategies have often been inadequate, in managing the residual muscle imbalance and muscle co-contraction. Therefore, we attempted to improve the functional movements, by using botulinum toxin type A, to reduce the abnormal co-contraction of the antagonist muscles.
机译:出生时臂丛神经麻痹(BBPP)通常是由分娩时的丛神经牵引引起的。尽管完全康复的可能性相对较高,但BBPP病例的5%至25%导致长期和持续的残疾。特别是,肩膀和肘部周围的肌肉失衡和共收缩会导致异常的运动表现,骨畸形和关节挛缩。物理和职业疗法最常用,但是这些常规的治疗策略在管理残留的肌肉失衡和肌肉共收缩方面常常是不充分的。因此,我们试图通过使用A型肉毒杆菌毒素来改善功能性运动,以减少拮抗肌的异常共收缩。

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