Cancer Stem Cells in Pancreatic Cancer

机译：胰腺癌中的癌症干细胞

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摘要

Pancreatic cancer is an aggressive malignant solid tumor well-known by early metastasis, local invasion, resistance to standard chemo- and radiotherapy and poor prognosis. Increasing evidence indicates that pancreatic cancer is initiated and propagated by cancer stem cells (CSCs). Here we review the current research results regarding CSCs in pancreatic cancer and discuss the different markers identifying pancreatic CSCs. This review will focus on metastasis, microRNA regulation and anti-CSC therapy in pancreatic cancer.

机译：胰腺癌是一种侵袭性恶性实体瘤，以早期转移，局部浸润，对标准化学和放射疗法的抵抗力以及预后不良而闻名。越来越多的证据表明，胰腺癌是由癌症干细胞（CSC）引发和繁殖的。在这里，我们审查有关胰腺癌的CSCs的当前研究结果，并讨论鉴定胰腺CSCs的不同标记。这篇综述将集中在胰腺癌的转移，microRNA调控和抗CSC治疗方面。

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期刊名称 Cancers
作者
Qi Bao; Yue Zhao; Andrea Renner; Hanno Niess; Hendrik Seeliger; Karl-Walter Jauch; Christiane J. Bruns;
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作者单位

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年(卷),期 2010(2),3
年度 2010
页码 1629–1641
总页数 13
原文格式 PDF
正文语种
中图分类肿瘤学;
关键词
cancer stem cells pancreatic cancer signaling pathways metastasis CSC-related therapy microRNAs;

机译：癌干细胞;胰腺癌;信号通路;转移;CSC相关疗法;microRNA;

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专利

1. Role of the Hypoxia-inducible factor-1 alpha induced autophagy in the conversion of non-stem pancreatic cancer cells into CD133+ pancreatic cancer stem-like cells [J] . Haitao Zhu, Dongqing Wang, Yanfang Liu, Cancer Cell International . 2013,第1期

机译：缺氧诱导因子-1α诱导的自噬在非干胰腺癌细胞向CD133 +胰腺干细胞转化中的作用
2. Role of the Hypoxia-inducible factor-1 alpha induced autophagy in the conversion of non-stem pancreatic cancer cells into CD133 + pancreatic cancer stem-like cells [J] . Haitao Zhu, Dongqing Wang, Yanfang Liu, Cancer Cell International . 2013,第1期

机译：缺氧诱导因子-1α诱导的自噬在非干胰腺癌细胞向CD133 +胰腺干细胞转化中的作用
3. Role of the Hypoxia-inducible factor-1 alpha induced autophagy in the conversion of non-stem pancreatic cancer cells into CD133+ pancreatic cancer stem-like cells [J] . Haitao Zhu, Dongqing Wang, Yanfang Liu, Cancer Cell International . 2013,第1期

机译：缺氧诱导因子-1α诱导的自噬在非干胰腺癌细胞向CD133 +胰腺干细胞转化中的作用
4. Enhancement of Effects of Chitosan and Nanoparticles on Pancreatic Cancer Cells by Combination Treatments with the Anti-Cancer Drug Adriamycin [C] . J.C.K.Lai, W.Gao, A.Bhushan TechConnect World Innovation Conference Expo;Annual SBIR/STTR Spring Innovation Conference;Annual Nanotech Conference Expo;AI TechConnect Spring Conference Expo . 2019

机译：用抗癌药物亚霉素组合治疗提高壳聚糖和纳米粒子对胰腺癌细胞的影响
5. NK cells lyse poorly differentiated but not well-differentiated pancreatic cancer cells; role of NK cells in selection and differentiation of pancreatic cancer stem cells. [D] . Maung, Phyu Ou. 2014

机译：NK细胞裂解分化差但分化差的胰腺癌细胞；细胞在胰腺癌干细胞选择和分化中的作用
6. Role of the Hypoxia-inducible factor-1 alpha induced autophagy in the conversion of non-stem pancreatic cancer cells into CD133+ pancreatic cancer stem-like cells [O] . Haitao Zhu, Dongqing Wang, Yanfang Liu, 2013

机译：缺氧诱导因子-1α诱导的自噬在非干胰腺癌细胞向CD133 +胰腺干细胞转化中的作用
7. Table 8.4.c.1 Infiltration of cancer cells at pancreatic cut end margin (PCM) Table 8.4.c.2 Procedure of pancreatectomy and PCM Table 8.4.c.3 Infiltration of cancer cells at bile duct cut end margin (BCM) Table 8.4.c.4 Infiltration of cancer cells at dissected pancreatic tissue margin (DPM) Table 8.4.c.5 Procedure of pancreatectomy and DPM Table 8.4.c.6 Residual tumor Table 8.4.c.7 Procedure of pancreatectomy and residual tumor Figure 8.4.c.8 Survival of patients with endocrine tumor after pancreatectomy according to the infiltration of cancer cells at pancreatic cut end margin (PCM) Figure 8.4.c.9 Survival of patients with endocrine tumor after pancreatectomy according to the infiltration of cancer cells at dissected pancreatic tissue margin (DPM) Figure 8.4.c.10 Survival of patients with endocrine tumor after pancreatectomy according to the residual tumor [O] . 2007

机译：表8.4.C.1胰腺切割端缘（PCM）癌细胞的浸润表8.4.C.2胰腺切除术和PCM的程序表8.4.C.3胆汁管道切口癌细胞浸润边缘（BCM）表8.4.c.4在解剖胰腺组织边缘（DPM）下癌细胞浸润表8.4.c.5胰腺切除术和DPM的程序表8.4.c.6残留的肿瘤表8.4 .c.7胰腺切除术和残留肿瘤的程序图8.4.c.8根据胰蛋白酶切除末端边缘（PCM）的癌细胞渗透后，胰腺切除术后的患者存活患者图8.4.c.9根据残留胰腺组织边缘的癌细胞渗透（DPM）渗透后，胰腺切除术后的患者存活患者图8.4.c.10根据残留肿瘤，胰腺切除术后的内分泌肿瘤患者存活

Cancer Stem Cells in Pancreatic Cancer

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