首页> 美国卫生研究院文献>Clinical Ophthalmology (Auckland N.Z.) >Membrane tissue on the optic disc may cause macular schisis associated with a glaucomatous optic disc without optic disc pits
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Membrane tissue on the optic disc may cause macular schisis associated with a glaucomatous optic disc without optic disc pits

机译:视盘上的膜组织可能会导致与青光眼视盘相关的黄斑裂孔而无视盘凹坑

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摘要

This article reports a case of possible involvement of membrane tissue on the optic disc with macular schisis formation associated with glaucomatous optic disc without optic disc pits. A 78-year-old man presented with loss of central visual acuity of the left eye. He had a medical history of primary open-angle glaucoma. Visual acuity was 0.6 in his left eye, and funduscopy revealed macular schisis and a glaucomatous optic disc without optic disc pits. Optical coherence tomography (OCT) revealed membrane tissue on the optic disc and a tunnel-like hyporeflective lesion connecting the schisis cavity and a site near the tissue, but no obvious optic disc pit. He underwent pars plana vitrectomy during which the membrane tissue on the disc and internal limiting membrane were removed. Posterior vitreous detachment was observed intraoperatively. Thereafter, the tunnel-like lesion observed on OCT was rapidly obscured and the macular schisis gradually reduced. Eighteen months after surgery, his visual acuity had improved to 0.9 with almost complete regression of the macular schisis. No optic disc pit was visualized after surgery. The changes in OCT findings described here suggest an etiology for macular schisis without optic disc pits in an eye with a glaucomatous optic disc with posterior vitreous detachment. A connection between the schisis cavity and the vitreous cavity may have appeared via the tunnel-like structure due to the membrane tissue exerting traction on the optic disc.
机译:本文报道一例可能累及视盘上的膜组织并伴有青光眼性视盘而无视盘凹坑的黄斑裂痕形成。一名78岁的男子表现出左眼中央视力丧失。他有原发性开角型青光眼的病史。他的左眼视力为0.6,眼底镜检查显示为黄斑裂痕和青光眼性视盘,无视盘凹坑。光学相干断层扫描(OCT)显示视盘上的膜组织和连接裂隙腔和组织附近部位的隧道状低反射性病变,但无明显的视盘凹坑。他接受了pars平面玻璃体切除术,在此期间,椎间盘和内部限制膜上的膜组织被切除。术中观察到玻璃体后脱离。此后,OCT上观察到的隧道样病变迅速被遮盖,黄斑裂痕逐渐减少。手术18个月后,他的视力提高到0.9,黄斑裂痕几乎完全消退。手术后未发现视盘凹坑。此处描述的OCT检查结果的变化提示黄斑裂孔病的病因是眼前具有玻璃体后脱离的青光眼性视盘而无视盘凹陷。由于膜组织在视盘上施加牵引力,因此可能通过隧道状结构在裂隙腔和玻璃体腔之间出现连接。

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