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MicroRNA-1296 inhibits metastasis and epithelial-mesenchymal transition of hepatocellular carcinoma by targeting SRPK1-mediated PI3K/AKT pathway

机译：MicroRNA-1296通过靶向SRPK1介导的PI3K / AKT途径抑制肝细胞癌的转移和上皮-间质转化

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BackgroundIncreasing evidences demonstrate that miRNAs contribute to development and progression of hepatocellular carcinoma (HCC). Underexpression of miR-1296 is recently reported to promote growth and metastasis of human cancers. However, the expression and role of miR-1296 in HCC remain unknown.

机译：背景技术越来越多的证据表明，miRNA有助于肝细胞癌（HCC）的发展和进程。最近报道，miR-1296的表达不足会促进人类癌症的生长和转移。但是，miR-1296在肝癌中的表达和作用仍然未知。

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期刊名称 Molecular Cancer
作者
Qiuran Xu; Xin Liu; Zhikui Liu; Zhenyu Zhou; Yufeng Wang; Jianfeng Tu; Lijie Li; Hangxing Bao; Liu Yang; Kangsheng Tu;
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作者单位

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年(卷),期 2017(16),-1
年度 2017
页码 103
总页数 15
原文格式 PDF
正文语种
中图分类分子生物学;肿瘤学;
关键词
MicroRNA-1296 Hepatocellular carcinoma SRPK1 Metastasis PI3K/AKT pathway;

机译：MicroRNA-1296;肝细胞癌;SRPK1;转移;PI3K / AKT途径;

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专利

1. MicroRNA-1296 inhibits metastasis and epithelial-mesenchymal transition of hepatocellular carcinoma by targeting SRPK1-mediated PI3K/AKT pathway [J] . Qiuran Xu, Xin Liu, Zhikui Liu, Molecular Cancer . 2017,第1期

机译：MicroRNA-1296通过靶向SRPK1介导的PI3K / AKT途径抑制肝细胞癌的转移和上皮-间质转化
2. miR-300 regulates the epithelial-mesenchymal transition and invasion of hepatocellular carcinoma by targeting the FAK/PI3K/AKT signaling pathway [J] . Wang Rongchang, Yu Zheng, Chen Fan, Biomedicine & pharmacotherapy =: Biomedecine & pharmacotherapie . 2018,第期

机译：MiR-300通过靶向FAK / PI3K / AKT信号通路来调节肝细胞癌的上皮 - 间充质转换和侵袭肝细胞癌
3. STYK1 promotes epithelial-mesenchymal transition and tumor metastasis in human hepatocellular carcinoma through MEK/ERK and PI3K/AKT signaling [J] . Zhaowen Wang, Lei Qu, Biao Deng, Scientific reports. . 2016,第1期

机译：STYK1促进通过MEK / ERK和PI3K / AKT信号传导上皮 - 间质转化和肿瘤转移在人肝癌
4. ROLE OF CD36 AND FREE FATTY ACID UPTAKE IN EPITHELIAL-MESENCHYMAL TRANSITION OF HEPATOCELLULAR CARCINOMA CELLS [C] . Christina Chan Biochemical and molecular engineering XX: the next generation of biochemical engineering: from nanoscale to industrial scale . 2017

机译：CD36和游离脂肪酸摄取在肝癌细胞上皮间质转化中的作用
5. The multi-targeted receptor tyrosine kinase inhibitor, linifanib (ABT-869), induces apoptosis and inhibits proliferation of leukemia cells through phosphoinositide-3 kinase (PI3K)/AKT-dependent signaling pathways. [D] . Hernandez, Jenny Elizabeth. 2010

机译：多靶点受体酪氨酸激酶抑制剂利尼法尼（ABT-869）通过磷酸肌醇3激酶（PI3K）/ AKT依赖性信号传导途径诱导凋亡并抑制白血病细胞的增殖。
6. c-Src activation promotes nasopharyngeal carcinoma metastasis by inducing the epithelial-mesenchymal transition via PI3K/Akt signaling pathway: a new and promising target for NPC [O] . Liangru Ke, Yanqun Xiang, Xiang Guo, 2016

机译：c-Src激活通过PI3K / Akt信号通路诱导上皮-间质转化促进鼻咽癌转移：NPC的新靶标
7. c-Src activation promotes nasopharyngeal carcinoma metastasis by inducing the epithelial-mesenchymal transition via PI3K/Akt signaling pathway: a new and promising target for NPC [O] . Liangru Ke, Yanqun Xiang, Xiang Guo, 2016

机译：通过PI3K / AKT信号通路诱导上皮间充质转化，C-SRC活化促进鼻咽癌转移：NPC的新和有前途的靶标

MicroRNA-1296 inhibits metastasis and epithelial-mesenchymal transition of hepatocellular carcinoma by targeting SRPK1-mediated PI3K/AKT pathway

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